Reduced adaptation of the pancreatic B cells during pregnancy is the major causal factor for gestational diabetes: Current knowledge and metabolic effects on the offspring

Acta Obstetricia et Gynecologica Scandinavica

Volumn 87, Issue 12, 2008, Pages 1266-1270

  Devlieger, Roland ^a   Casteels, Kristina ^a   Van Assche, Frans A ^a,b  

^a UNIVERSITY HOSPITALS LEUVEN   (Belgium)

^b UNIVERSITY OF LEUVEN   (Belgium)

Author keywords

Gestational diabetes; Offspring; Pregnancy

Indexed keywords

11BETA HYDROXYSTEROID DEHYDROGENASE; GLUCOKINASE; GLUCOSE TRANSPORTER 1; GLUCOSE TRANSPORTER 4; INSULIN; METHIONINE; STREPTOZOCIN;

CELL FUNCTION; CELL PROLIFERATION; CELL STRUCTURE; FETUS RISK; GENE MUTATION; GENETIC PREDISPOSITION; GLUCOSE METABOLISM; GLUCOSE TOLERANCE; HUMAN; HYPERGLYCEMIA; INSULIN DEPENDENT DIABETES MELLITUS; INSULIN RELEASE; INSULIN RESISTANCE; LACTATION; MATERNAL DIABETES MELLITUS; METABOLIC SYNDROME X; NON INSULIN DEPENDENT DIABETES MELLITUS; NONHUMAN; NOTE; PANCREAS ISLET BETA CELL; PATHOGENESIS; PREGNANCY DIABETES MELLITUS; PRIORITY JOURNAL; PROGENY;

ADAPTATION, PHYSIOLOGICAL; ANIMALS; BLOOD GLUCOSE; DIABETES, GESTATIONAL; DISEASE MODELS, ANIMAL; FEMALE; HUMANS; INSULIN; INSULIN RESISTANCE; INSULIN-SECRETING CELLS; PREGNANCY;

EID: 57449100729     PISSN: 00016349     EISSN: 16000412     Source Type: Journal    
DOI: 10.1080/00016340802443863     Document Type: Note

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