Estimation of relation between homocysteine concentration and selected lipid parameters and adhesion molecules concentration in children with atherosclerosis risk factors;Ocena zależności pomiȩdzy stȩżeniem homocysteiny a stȩżeniem wybranych parametrów przemiany lipidowej i cza̧steczek adhezyjnych u dzieci z czynnikami ryzyka miażdżycy

Polski Merkuriusz Lekarski

Volumn 25, Issue 148, 2008, Pages 356-360

  Sierakowska Fijałek, Anna ^a   Baj, Zbigniew ^a   Kaczmarek, Piotr ^a   Stȩpień, Mariusz ^a   Rysz, Jacek ^a  

^a MEDICAL UNIVERSITY OF LODZ   (Poland)

Author keywords

Adhesion molecules; Atherosclerosis; Children; Homocysteine; Lipid levels

Indexed keywords

BIOLOGICAL MARKER; CELL ADHESION MOLECULE; HOMOCYSTEINE; SELECTIN; VASCULAR CELL ADHESION MOLECULE 1;

ADOLESCENT; ARTICLE; ATHEROSCLEROSIS; BLOOD; COMORBIDITY; DISORDERS OF LIPID AND LIPOPROTEIN METABOLISM; FEMALE; HUMAN; HYPERHOMOCYSTEINEMIA; HYPERTENSION; LIPID METABOLISM; MALE; METABOLISM; OBESITY; RISK FACTOR;

ADOLESCENT; ATHEROSCLEROSIS; BIOLOGICAL MARKERS; CELL ADHESION MOLECULES; COMORBIDITY; FEMALE; HOMOCYSTEINE; HUMANS; HYPERHOMOCYSTEINEMIA; HYPERTENSION; LIPID METABOLISM; LIPID METABOLISM DISORDERS; MALE; OBESITY; RISK FACTORS; SELECTINS; VASCULAR CELL ADHESION MOLECULE-1;

EID: 56249110978     PISSN: 14269686     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (25)

1. Austin R.C, Lentz S.R., Werstuck G.H.: Role of hyperhomocysteinemia in endothelial dysfunction and atherothrombotic disease. Cell. Death Differ., 2004, 11, suppl. 1, S 56-64.
2. Berenson G.S.: Childhood risk factors predict adult risk associated with subclinical cardiovascular disease. The Bogalusa Heart Study. Am. J. Cardiol., 2002, 21, 90, 3L-7L.
3. Binder C.J., Chang M.K., Shaw P.X. i wsp.: Innaate and acquired immunity in atherogenesis Nature Medicine, 2002, 11, 1218-1226.
4. Blake G.J., Ridker P.M.: Novel Clinical Markers of Vascular Wall Inflammation. Circ. Res., 2001, 89, 763-771.
5. Blann A.D., Nadar S.K., Lip G.Y.: The adhesion molecule P-selectin and cardiovascular disease. European Heart Journal, 2003, 24, 2166-2179.
6. Castro R., Rivera I., Blom H.J. i wsp.: Homocysteine metabolism, hyperhomocysteinaemia and vascular disease: an overview. J. Inherit. Metab. Dis. 2006, 29, 1, 3-20.
7. Głowińska-Olszewska B., Tołwińska J., Urban M.: Relationship between endothelial dysfunction, carotid artery intima media thickness and circulating markers of vascular inflammation in obese hypertensive children and adolescents. J. Pediatr. Endocrinol. Metab. 2007, 20, 10, 1125-1136.
8. Guthiconda S., Haynes W.G.: Homocysteine: role and implications in atherosclerosis. Curr. Atheroscler. Rep. 2006, 8, 2, 100-106.
9. Jakubowski H.: Pathophysiological consequences of homocysteine excess. J. Nutr. 2006, 136, 6 suppl. 1741S-1749S.
10. Kavazarakis E., Moustaki M., Gourgiotis D. i wsp: The impact of serum lipid levels on circulating soluble adhesion molecules in childhood. Pediatr. Res., 2002, 52, 3, 454-458.
11. Kulikowska-Karpińska E., Moniuszko-Jakoniuk J.: The antioxidant barrier in the organism. Pol. J. Environ. Stud. 2004, 13, 5-13.
12. Lentz S. R.: Mechanismus of homocysteine - induced atherothrombosis. J. Thromb. Haemost. 2005, 3, 1646-1654.
13. Li S., Chen W., Srinivasan S.R. i wsp.: Childhood cardiovascular risk factors and carotid vascular changes in adulthood. JAMA, 2003, 290, 2271-2276.
14. McCully K.: Vascular pathology of homocysteinemia: implication for pathogenesis of atherosclerosis. Am. J. Pathol., 1969, 56, 111-112.
15. Musial K., Zwolińska D.: Adhesion molecules, cytokines and endothelial dysfunction in atherosclerosis. Adv. Clin. Exp. Med., 2006, 15, 6, 971-978.
16. National Cholesterol Education Programme. Raport of the Expert Panel on Blood Cholesterol Levels in Children and Adolescents. Pediatrics, 1992, 89, suppl., 525-584.
17. Ohta T., Saku K., Takata K. i wsp.: Soluble vascular cell adhesion molecule-1 and soluble intercellular adhesion molecule-1 correlate with lipid and apolipoprotein risk factors for coronary artery disease in children. Eur. J. Pediatr., 1999, 158, 7, 592-598.
18. Packard R.R., Libby P.: Inflammation in atherosclerosis: from vascular biology to biomarker discovery and risk prediction. Clin. Chem. 2008, 54, 1, 24-38.
19. Poredos P.: Endothelial dysfunction in the pathogenesis of atherosclerosis. Int. Angiol., 2002, 2, 109-116.
20. Ross R.: Atherosclerosis an inflammation disease. N. Engl. J. Med., 1999, 340, 115-126.
21. Szymczak E., Chetchowska M., Radomyska B. i wsp.: Homocysteins i wybrane parametry przemian lipidowych u dzieci z hipercholesterolemia̧ . Med. Wieku Rozw., 2001, 5, 2, 157-164.
22. Tonstad S., Refsum H., Keland P.: Association between plasma total homocysteine and parental history of cardiovascular disease in children with familial hypercholesterolemia. Circulation, 1997, 96, 1803-1808.
23. Williams CL., Hayman L.L., Daniels S.R. i wsp.: Cardiovascular health in childhood: a statement for health professionals from the Committee on Atherosclerosis, Hypertension, and Obesity in the Young (AHOY) of the Council on Cardiovascular Disease in the Young, American Heart Association. Circulation, 2002, 106-143.
24. Włodarska M., Gawor Z., Piotrowski G. i wsp.: Ocena czynników ryzyka choroby niedokrwiennej serca u studentów Politechniki Czȩstochowskiej z otyłościa̧ i nadwaga̧. Czynniki Ryzyka, 2004, 1-2, 23-31.
25. Zapolska-Downar D.: Dysfunkcja komórek śró dbłonka jako jeden z czynników patogenetycznych miażdżycy. Normalizuja̧cy wpływ niektórych leków. Czynniki Ryzyka 2000, 4, 5-15.