Apoptosis of human pulmonary adenocarcinoma cells mediated by arsenic trioxide (As2O3) via increasing intracellular calcium concentration

Chinese Pharmacological Bulletin

Volumn 20, Issue 8, 2004, Pages 867-870

  Zhang, Li ^a   Wang, Ling ^a   Song, Wei Hua ^a   Wen, Wei ^a   Sun, Hong Li ^a   Wu, Zhao Ju ^a   Yang, Bao Feng ^a,b  

^a HARBIN MEDICAL UNIVERSITY   (China)

^b State Province Key Laboratory of Biopharmaceutical Engineering   (China)

Author keywords

AGZY 83 a; Apoptosis; As2O3; Calcium

Indexed keywords

ARSENIC TRIOXIDE;

ANTINEOPLASTIC ACTIVITY; APOPTOSIS; ARTICLE; CALCIUM CELL LEVEL; CANCER CELL; CANCER INHIBITION; CELL NUCLEUS; CELL PROLIFERATION; CONCENTRATION RESPONSE; CONTROLLED STUDY; DRUG ACTIVITY; DRUG EFFECT; DRUG MECHANISM; FLOW CYTOMETRY; HUMAN; HUMAN CELL; LASER MICROSCOPY; LUNG ADENOCARCINOMA; TRANSMISSION ELECTRON MICROSCOPY;

EID: 5044238920     PISSN: 10011978     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (7)

1. Bast RC, Kule DW, Pollock RE et al. Holland-Frei Cancer Medicine[M], 5th Ed Bc Decker, chap, 2000: 120.
2. Wilson H, Hyman M, Janet S et al. Mechanisms of action of Arsenic Trioxide[J]. Cancer Res, 2002, 62(14): 3893-903.