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Volumn 1, Issue 2, 2008, Pages 222-238
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Do pharmacological approaches that prevent opioid tolerance target different elements in the same regulatory machinery?
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Author keywords
[No Author keywords available]
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Indexed keywords
CALCIUM CALMODULIN DEPENDENT PROTEIN KINASE II;
CYCLIC AMP DEPENDENT PROTEIN KINASE;
DIAMORPHINE;
G PROTEIN COUPLED RECEPTOR;
G PROTEIN COUPLED RECEPTOR KINASE;
MORPHINE;
MU OPIATE RECEPTOR;
N METHYL DEXTRO ASPARTIC ACID RECEPTOR;
NARCOTIC AGENT;
PROTEIN KINASE C;
RGS PROTEIN;
ADULT;
ANIMAL;
BRAIN;
DOSE RESPONSE;
DRUG EFFECT;
DRUG TOLERANCE;
HEROIN DEPENDENCE;
HUMAN;
MORPHINE ADDICTION;
MOUSE;
PATHOPHYSIOLOGY;
PHYSIOLOGY;
REVIEW;
SIGNAL TRANSDUCTION;
WITHDRAWAL SYNDROME;
ANIMALS;
BRAIN;
CALCIUM-CALMODULIN-DEPENDENT PROTEIN KINASE TYPE 2;
CYCLIC AMP-DEPENDENT PROTEIN KINASES;
DOSE-RESPONSE RELATIONSHIP, DRUG;
DRUG TOLERANCE;
G-PROTEIN-COUPLED RECEPTOR KINASES;
HEROIN;
HEROIN DEPENDENCE;
HUMANS;
MICE;
MORPHINE;
MORPHINE DEPENDENCE;
NARCOTICS;
PROTEIN KINASE C;
RECEPTORS, G-PROTEIN-COUPLED;
RECEPTORS, N-METHYL-D-ASPARTATE;
RECEPTORS, OPIOID, MU;
RGS PROTEINS;
SIGNAL TRANSDUCTION;
SUBSTANCE WITHDRAWAL SYNDROME;
YOUNG ADULT;
MLCS;
MLOWN;
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EID: 49549084688
PISSN: None
EISSN: 18744745
Source Type: Journal
DOI: 10.2174/1874473710801020222 Document Type: Review |
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Times cited : (49)
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References (207)
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