|
Volumn 18, Issue 7, 2004, Pages 857-859
|
Disruption of Rho signaling results in progressive atrioventricular conduction defects while ventricular function remains preserved.
a a a a a a a a a a a a a a |
Author keywords
[No Author keywords available]
|
Indexed keywords
CONNEXIN 40;
CONNEXIN 43;
GAP JUNCTION PROTEIN;
GUANINE NUCLEOTIDE DISSOCIATION INHIBITOR;
MYOSIN HEAVY CHAIN;
RHO GUANINE NUCLEOTIDE BINDING PROTEIN;
RHO GUANINE NUCLEOTIDE DISSOCIATION INHIBITORS;
ADAPTATION;
ANIMAL;
ARTICLE;
BIOSYNTHESIS;
CATTLE;
CONGENITAL MALFORMATION;
DISEASE COURSE;
DRUG ANTAGONISM;
ENZYMOLOGY;
GENE EXPRESSION REGULATION;
GENETICS;
HEART ATRIOVENTRICULAR NODE;
HEART ATRIUM;
HEART BLOCK;
HEART LEFT VENTRICLE FUNCTION;
HEART MUSCLE CELL;
HETEROZYGOTE;
HYPERTROPHIC CARDIOMYOPATHY;
METABOLISM;
MOUSE;
PATHOPHYSIOLOGY;
PHYSIOLOGY;
PROMOTER REGION;
SIGNAL TRANSDUCTION;
TRANSGENE;
TRANSGENIC MOUSE;
ADAPTATION, PHYSIOLOGICAL;
ANIMALS;
ATRIOVENTRICULAR NODE;
CARDIOMYOPATHY, HYPERTROPHIC;
CATTLE;
CONNEXIN 43;
CONNEXINS;
DISEASE PROGRESSION;
GENE EXPRESSION REGULATION;
GUANINE NUCLEOTIDE DISSOCIATION INHIBITORS;
HEART ATRIA;
HEART BLOCK;
HETEROZYGOTE;
MICE;
MICE, TRANSGENIC;
MYOCYTES, CARDIAC;
MYOSIN HEAVY CHAINS;
PROMOTER REGIONS (GENETICS);
RHO GTP-BINDING PROTEINS;
SIGNAL TRANSDUCTION;
TRANSGENES;
VENTRICULAR FUNCTION, LEFT;
|
EID: 4644288506
PISSN: None
EISSN: 15306860
Source Type: Journal
DOI: 10.1096/fj.03-0664fje Document Type: Article |
Times cited : (41)
|
References (0)
|