Inactivation of Gi proteins by pertussis toxin diminishes the effectiveness of adrenergic stimuli in conduit arteries from spontaneously hypertensive rats

Physiological Research

Volumn 57, Issue 2, 2008, Pages 299-302

  Zemancikova A ^a   Torok J ^a   Zicha, J ^b   Kunes J ^b  

^a INSTITUTE OF CHEMISTRY   (Slovakia)

^b INSTITUTE OF PHYSIOLOGY   (Czech Republic)

Author keywords

Adrenergic contractions; Angiotensin II; Conduit arteries; Gi proteins; Hypertension; Pertussis toxin

Indexed keywords

ACETYLCHOLINE; ADRENERGIC RECEPTOR STIMULATING AGENT; ANGIOTENSIN II; ENZYME INHIBITOR; INHIBITORY GUANINE NUCLEOTIDE BINDING PROTEIN; NORADRENALIN; PERTUSSIS TOXIN;

ANIMAL; ARTERY; ARTICLE; BLOOD PRESSURE; DRUG ANTAGONISM; DRUG EFFECT; ELECTROSTIMULATION; HYPERTENSION; IN VITRO STUDY; MALE; METABOLISM; PHYSIOLOGY; PULMONARY ARTERY; RAT; SIGNAL TRANSDUCTION; SPONTANEOUSLY HYPERTENSIVE RAT; SUPERIOR MESENTERIC ARTERY; THORACIC AORTA; VASCULAR ENDOTHELIUM;

ACETYLCHOLINE; ADRENERGIC AGENTS; ANGIOTENSIN II; ANIMALS; AORTA, THORACIC; ARTERIES; BLOOD PRESSURE; ELECTRIC STIMULATION; ENDOTHELIUM, VASCULAR; ENZYME INHIBITORS; GTP-BINDING PROTEIN ALPHA SUBUNITS, GI-GO; HYPERTENSION; MALE; MESENTERIC ARTERY, SUPERIOR; NOREPINEPHRINE; PERTUSSIS TOXIN; PULMONARY ARTERY; RATS; RATS, INBRED SHR; SIGNAL TRANSDUCTION;

EID: 45849128377     PISSN: 08628408     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (16)

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