Iron and hepatitis C: What can we learn from thalassemia major?

Liver International

Volumn 28, Issue 6, 2008, Pages 892-893

  Mahmoud, Amira ^a   Musallam, Khaled M ^b   Taher, Ali T ^b  

^a MAFRAQ HOSPITAL   (United Arab Emirates)

^b AMERICAN UNIVERSITY OF BEIRUT   (Lebanon)

Author keywords

[No Author keywords available]

Indexed keywords

ALANINE AMINOTRANSFERASE; DEFERASIROX; DEFEROXAMINE; FERRITIN; IRON; IRON CHELATING AGENT;

ADULT; ALANINE AMINOTRANSFERASE BLOOD LEVEL; BLOOD TRANSFUSION; CASE REPORT; DRUG SUBSTITUTION; DRUG WITHDRAWAL; FEMALE; FERRITIN BLOOD LEVEL; HEPATITIS C; HEPATITIS C VIRUS; HUMAN; IRON DEFICIENCY; IRON METABOLISM; IRON OVERLOAD; IRON STORAGE; LETTER; MALE; POLYMERASE CHAIN REACTION; THALASSEMIA MAJOR;

ADULT; BENZOIC ACIDS; BETA-THALASSEMIA; CHELATION THERAPY; DEFEROXAMINE; FEMALE; FERRITINS; HEPACIVIRUS; HEPATITIS C, CHRONIC; HUMANS; IRON; MALE; PHLEBOTOMY; SIDEROPHORES; TRIAZOLES;

EID: 44949117041     PISSN: 14783223     EISSN: 14783231     Source Type: Journal    
DOI: 10.1111/j.1478-3231.2008.01769.x     Document Type: Letter

References (5)

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5. Tsai NC, Zuckerman E, Han SH, Goad K, Redeker AG, Fong TL. Effect of iron depletion on long-term response to interferon-alpha in patients with chronic hepatitis C who previously did not respond to interferon therapy. Am J Gastroenterol 1997; 92: 1831-4.