Is the metabolic syndrome a disease of the foregut? Yes, excessive foregut

Annals of Surgery

Volumn 247, Issue 6, 2008, Pages 1074-1075

  Santoro, Sergio ^a  

^a HOSPITAL ISRAELITA ALBERT EINSTEIN   (Brazil)

Author keywords

[No Author keywords available]

Indexed keywords

ABDOMEN; ANATOMY; BILIOPANCREATIC BYPASS; DIETARY INTAKE; GASTROINTESTINAL DISEASE; GASTROINTESTINAL TRACT; GASTROINTESTINAL TRACT FUNCTION; HUMAN; INTERMETHOD COMPARISON; INTESTINE SURGERY; JEJUNECTOMY; LETTER; MALABSORPTION; METABOLIC SYNDROME X; MORTALITY; NON INSULIN DEPENDENT DIABETES MELLITUS; NONHUMAN; OBESITY; OMENTECTOMY; PHYSICAL ANTHROPOLOGY; PRIORITY JOURNAL; ROUX Y ANASTOMOSIS; SLEEVE GASTRECTOMY; SURGICAL TECHNIQUE; WAIST HIP RATIO;

HUMANS; INTESTINE, SMALL; METABOLIC SYNDROME X;

EID: 44449127705     PISSN: 00034932     EISSN: None     Source Type: Journal    
DOI: 10.1097/SLA.0b013e3181758ddb     Document Type: Letter

References (17)

1. Hickey MS, Pories WJ, MacDonald KG Jr, et al. A new paradigm for type 2 diabetes mellitus: could it be a disease of the foregut? Ann Surg. 1998;227:637-643.
2. Lam NT, Kieffer TJ. The multifaceted potential of glucagon-like peptide-1 as a therapeutic agent. Minerva Endocrinol. 2002;27:79-93.
3. Meier JJ, Gethmann A, Götze O, et al. Glucagon-like peptide 1 abolishes the postprandial rise in triglyceride concentrations and lowers levels of non-esterified fatty acids in humans. Diabetologia. 2006;49:452-458.
4. Ranganath LR, Beety JM, Morgan LM, et al. Attenuated GLP-1 secretion in obesity: cause or consequence? Gut. 1996;38:916-919.
5. Lugari R, Dei Cas A, Ugolotti D, et al. Evidence for early impairment of glucagon-like peptide 1-induced insulin secretion in human type 2 (non insulin-dependent) diabetes. Horm Metab Res. 2002;34:150-154.
6. Grundy SM, Brewer HB Jr, Cleeman JI, et al. Definition of metabolic syndrome. Report of the National Heart, Lung, and Blood Institute/American Heart Association conference on scientific issues related to definition. Circulation. 2004;109:433-438.
7. Milton K. The critical role played by animal source foods in human (Homo) evolution. J Nutr. 2003;133:3886S-3892S.
8. Buchwald H, Avidor Y, Braunwald E, et al. Bariatric surgery: a systematic review and meta-analysis. JAMA. 2004;292:1724-1737.
9. Ulshen MH, Hoyt EC, Fuller CR, et al. Increased ileal proglucagon expression after jejunectomy is not suppressed by inhibition of bowel growth. Dig Dis Sci. 1996;41:677-683.
10. Stevens CE, Hume ID. Comparative Physiology of the Vertebrate Digestive System. 2nd ed. Cambridge: Cambridge University Press; 1995.
11. Leonard WR, Robertson ML. Evolutionary perspectives on human nutrition: the influence of brain and body size on diet and metabolism. Am J Hum Biol. 1994;6:77-88.
12. Aiello LC, Wheeler P. The expensive tissue hypothesis: the brain and the digestive system in human and primate evolution. Curr Anthropol. 1995;36:199-221.
13. Hounnou G, Destrieux C, Desme J, et al. Anatomical study of the length of the human intestine. Surg Radiol Anat. 2002;24:290-294.
14. Santoro S. Hipertrofia intestinal induzida por alimento e obesidade. Einstein. 2005;3:310-312.
15. Santoro S, Milleo FQ, Malzoni CE, et al. Enterohormonal changes after digestive adaptation: five-year results of a surgical proposal to treat obesity and associated diseases. Obes Surg. 2008;18:17-26.
16. Rubino F, Marescaux J. Effect of duodenaljejunal exclusion in a non-obese animal model of type 2 diabetes: a new perspective for an old disease. Ann Surg. 2004;239:1-11.
17. Santoro S, Malzoni CE, Velhote MCP, et al. Digestive adaptation with intestinal reserve: a neuroendocrine-based procedure for morbid obesity. Obes Surg. 2006;16:1371-1379.