Prominent pancreatic endocrinopathy and altered control of food intake disrupt energy homeostasis in prion diseases

Journal of Endocrinology

Volumn 197, Issue 2, 2008, Pages 251-263

  Bailey, J D ^a   Berardinelli, J G ^a   Rocke, T E ^b   Bessen, R A ^a  

^a MONTANA STATE UNIVERSITY   (United States)

^b NATIONAL WILDLIFE HEALTH CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GLUCAGON; GLUCOSE; INSULIN; KETONE DERIVATIVE; LEPTIN; PRION PROTEIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ENERGY BALANCE; FLUID INTAKE; FOOD INTAKE; GLUCAGON BLOOD LEVEL; GLUCAGON RELEASE; GLUCOSE BLOOD LEVEL; HAMSTER; HOMEOSTASIS; HYPERGLUCAGONEMIA; HYPERGLYCEMIA; HYPERINSULINEMIA; HYPERLEPTINEMIA; HYPERPHAGIA; HYPOGLYCEMIA; INSULIN BLOOD LEVEL; MALE; NONHUMAN; PANCREAS ISLET DISEASE; POLYDIPSIA; PRION DISEASE; PRIORITY JOURNAL; PROTEIN BLOOD LEVEL; SATIETY; SCRAPIE AGENT; STRAIN DIFFERENCE; TRANSMISSIBLE MINK ENCEPHALOPATHY AGENT; VIRUS STRAIN; WEIGHT GAIN; WEIGHT REDUCTION;

ANIMALS; BLOOD GLUCOSE; BODY WEIGHT; CRICETINAE; DRINKING; EATING; ENERGY METABOLISM; GLUCAGON; GLUCOSE TOLERANCE TEST; HOMEOSTASIS; HYPOTHALAMUS; INSULIN; LEPTIN; MALE; MESOCRICETUS; PANCREATIC DISEASES; PRION DISEASES; PRPSC PROTEINS;

EID: 43849085313     PISSN: 00220795     EISSN: 14796805     Source Type: Journal    
DOI: 10.1677/JOE-07-0516     Document Type: Article

References (55)