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Volumn 26, Issue 13, 2008, Pages 2228-2230
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KRAS mutation signature in colorectal tumors significantly overlaps with the cetuximab response signature
a
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Author keywords
[No Author keywords available]
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Indexed keywords
B RAF KINASE;
CETUXIMAB;
DNA;
K RAS PROTEIN;
RNA;
ANTINEOPLASTIC AGENT;
EGFR PROTEIN, HUMAN;
EPIDERMAL GROWTH FACTOR RECEPTOR;
KRAS PROTEIN, HUMAN;
MONOCLONAL ANTIBODY;
ONCOPROTEIN;
RAS PROTEIN;
CANCER CHEMOTHERAPY;
CANCER STAGING;
COLORECTAL TUMOR;
DNA EXTRACTION;
GENE MUTATION;
GENE SEQUENCE;
LETTER;
MONOTHERAPY;
ONCOGENE K RAS;
PRIORITY JOURNAL;
RNA EXTRACTION;
SEQUENCE HOMOLOGY;
TREATMENT RESPONSE;
CLUSTER ANALYSIS;
DNA MICROARRAY;
DRUG ANTAGONISM;
GENE EXPRESSION PROFILING;
GENE EXPRESSION REGULATION;
GENETICS;
HUMAN;
METABOLISM;
METHODOLOGY;
MUTATION;
NOTE;
PATHOLOGY;
PATIENT SELECTION;
TREATMENT OUTCOME;
ANTIBODIES, MONOCLONAL;
ANTINEOPLASTIC AGENTS;
CLUSTER ANALYSIS;
COLORECTAL NEOPLASMS;
GENE EXPRESSION PROFILING;
GENE EXPRESSION REGULATION, NEOPLASTIC;
HUMANS;
MUTATION;
NEOPLASM STAGING;
OLIGONUCLEOTIDE ARRAY SEQUENCE ANALYSIS;
PATIENT SELECTION;
PROTO-ONCOGENE PROTEINS;
RAS PROTEINS;
RECEPTOR, EPIDERMAL GROWTH FACTOR;
TREATMENT OUTCOME;
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EID: 43749088703
PISSN: 0732183X
EISSN: None
Source Type: Journal
DOI: 10.1200/JCO.2007.15.9186 Document Type: Letter |
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Times cited : (32)
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References (4)
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