Time course of necrosis/apoptosis and neovascularization during experimental gastric conditioning

Diseases of the Esophagus

Volumn 21, Issue 4, 2008, Pages 370-376

  Lamas, S ^a   Azuara, D ^a   de Oca, Javier ^a,b   Sans, M ^a   Farran, L ^a   Alba, E ^a   Escalante, E ^a   Rafecas, A ^a  

^a HOSPITAL UNIVERSITARI DE BELLVITGE   (Spain)

^b UNIVERSITY OF BARCELONA   (Spain)

Author keywords

Apoptosis; Caspase 3; Delay phenomenon; Gastric conditioning; Necrosis; Neovascularization

Indexed keywords

CASPASE 3; DNA FRAGMENT; KI 67 ANTIGEN;

ANGIOGENESIS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BLOOD VESSEL; CELL DEATH; CELL PROLIFERATION; CONTROLLED STUDY; DEVASCULARIZATION; ENDOTHELIUM CELL; ENZYME ACTIVITY; ESOPHAGUS ANASTOMOSIS; ESOPHAGUS RESECTION; EXPERIMENTAL MODEL; HISTOPATHOLOGY; INTESTINE ISCHEMIA; ISCHEMIC PRECONDITIONING; MALE; NEOVASCULARIZATION (PATHOLOGY); NERVE LIGATION; NONHUMAN; PRIORITY JOURNAL; RAT; STOMACH FUNDUS; STOMACH TRANSPOSITION; TIME; TISSUE INJURY; VASCULAR ENDOTHELIUM;

ANIMALS; APOPTOSIS; DISEASE MODELS, ANIMAL; ESOPHAGUS; ISCHEMIC PRECONDITIONING; MALE; NECROSIS; NEOVASCULARIZATION, PATHOLOGIC; RATS; RATS, SPRAGUE-DAWLEY; STOMACH;

EID: 43449103950     PISSN: 11208694     EISSN: 14422050     Source Type: Journal    
DOI: 10.1111/j.1442-2050.2007.00772.x     Document Type: Article

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