Antibodies and other biomarkers - Pathological consequences (2)

Lupus

Volumn 17, Issue 3, 2008, Pages 256-258

  Ehrenstein, Michael R ^a   Alves, J D ^b  

^a UNIVERSITY COLLEGE LONDON   (United Kingdom)

^b UNIVERSIDADE NOVA DE LISBOA   (Portugal)

Author keywords

[No Author keywords available]

Indexed keywords

APOLIPOPROTEIN A1; ARYLDIALKYLPHOSPHATASE; AUTOANTIBODY; BIOLOGICAL MARKER;

CONFERENCE PAPER; FCGR3B GENE; GENE; GENE DELETION; GENETIC ASSOCIATION; GENETIC POLYMORPHISM; IMMUNOPATHOGENESIS; PATHOPHYSIOLOGY; PRIORITY JOURNAL; SYSTEMIC LUPUS ERYTHEMATOSUS;

ANIMALS; AUTOANTIBODIES; B-LYMPHOCYTES; BIOLOGICAL MARKERS; GENETIC PREDISPOSITION TO DISEASE; HUMANS; LIPOPROTEINS, HDL; LUPUS ERYTHEMATOSUS, SYSTEMIC; RECEPTORS, IGG;

EID: 42549139381     PISSN: 09612033     EISSN: None     Source Type: Journal    
DOI: 10.1177/0961203307088253     Document Type: Conference Paper

References (8)

1. Alves, JD, Grima, B. Oxidative stress in systemic lupus erythematosus and antiphospholipid syndrome: a gateway to atherosclerosis. Curr Rheumatol Rep 2003; 5: 383-390.
2. Delgado Alves, J, Kumar, S, Isenberg, DA. Cross-reactivity between anti-cardiolipin, anti-high-density lipoprotein and anti-apolipoprotein A-I IgG antibodies in patients with systemic lupus erythematosus and primary antiphospholipid syndrome. Rheumatology (Oxford) 2003; 42: 893-899.
3. Martin, F, Chan, AC. B cell immunobiology in disease: evolving concepts from the clinic. Annu Rev Immunol 2006; 24: 467-496.
4. Jacobi, AM, Odendahl, M, Reiter, K, et al. Correlation between circulating CD27high plasma cells and disease activity in patients with systemic lupus erythematosus. Arthritis Rheum 2003; 48: 1332-1342.
5. Mauri, C, Gray, D, Mushtaq, N, Londei, M. Prevention of arthritis by interleukin 10-producing B cells. J Exp Med 2003; 197: 489-501.
6. Evans, JG, Chavez-Rueda, KA, Eddaoudi, A, et al. Novel suppressive function of transitional 2 B cells in experimental arthritis. J Immunol 2007; 178: 7868-7878.
7. Aitman, TJ, Dong, R, Vyse, TJ, et al. Copy number polymorphism in Fcgr3 predisposes to glomerulonephritis in rats and humans. Nature 2006; 439: 851-855.
8. Fanciulli, M, Norsworthy, PJ, Petretto, P, et al. FCGR3B copy number variation is associated with susceptibility to systemic but not organ-specific autoimmunity. Nat Genet 2007; 39: 721-723.