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Volumn 6, Issue 9, 2007, Pages 1341-1350
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Cell cycle arrest by glucocorticoids may protect normal tissue and solid tumors from cancer therapy
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Author keywords
Apoptosis; Cancer therapy; Cell cycle; Dexamethasone; Glucocorticoid receptor; Glucocorticoids; Solid tumors
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Indexed keywords
CAMPTOTHECIN;
CARBOPLATIN;
CARMUSTINE;
CISPLATIN;
CORTISONE;
CYCLOPHOSPHAMIDE;
CYTARABINE;
DACTINOMYCIN;
DEXAMETHASONE;
DOXORUBICIN;
EPIRUBICIN;
ETOPOSIDE;
FLUOROURACIL;
GEMCITABINE;
GLUCOCORTICOID;
GLUCOCORTICOID RECEPTOR;
IFOSFAMIDE;
MERCAPTOPURINE;
METHOTREXATE;
MIFEPRISTONE;
NIMUSTINE;
PACLITAXEL;
PREDNISONE;
STAUROSPORINE;
TAMOXIFEN;
TENIPOSIDE;
TIOGUANINE;
UNINDEXED DRUG;
VINBLASTINE;
VINCRISTINE;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
APOPTOSIS;
BRAIN CANCER;
BREAST CANCER;
CANCER RADIOTHERAPY;
CANCER RESISTANCE;
CANCER THERAPY;
CELL CYCLE ARREST;
CELL CYCLE PROGRESSION;
CELL PROTECTION;
CELL SURVIVAL;
CLINICAL TRIAL;
COLON CANCER;
DRUG POTENTIATION;
GASTRITIS;
HUMAN;
HUMAN CELL;
LEWIS CARCINOMA;
LYMPHOID CELL;
MOUSE;
MULTIPLE CYCLE TREATMENT;
NAUSEA;
NEUROLOGIC DISEASE;
NONHUMAN;
OVARY CANCER;
PROSTATE CANCER;
RAT;
REVIEW;
SOFT TISSUE SARCOMA;
SOLID TUMOR;
THROMBOCYTOPENIA;
TOXICITY;
UTERINE CERVIX CANCER;
VOMITING;
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EID: 42549108158
PISSN: 15384047
EISSN: 15558576
Source Type: Journal
DOI: 10.4161/cbt.6.9.4765 Document Type: Review |
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Times cited : (61)
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References (112)
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