Signalling mechanisms and oxidative stress in apoptosis

Toxicology Letters

Volumn 82-83, Issue C, 1995, Pages 149-153

  Slater, Andrew F G ^a   Stefan, C ^a   Nobel, I ^a   Van Den Dobbelsteen, Diels J ^a   Orrenius, sten ^a  

^a KAROLINSKA INSTITUTE   (Sweden)

Author keywords

Dithiocarbamate; Glutathione; ICE like proteases; Necrosis; ROS; Thymocyte

Indexed keywords

ALKYLATING AGENT; BUTHIONINE SULFOXIMINE; DITHIOCARBAMIC ACID DERIVATIVE; ENZYME INHIBITOR; ETOPOSIDE; GLUCOCORTICOID; GLUTATHIONE; HYDROGEN PEROXIDE; PROTEINASE; QUINONE DERIVATIVE; REACTIVE OXYGEN METABOLITE;

ANIMAL CELL; APOPTOSIS; CONFERENCE PAPER; HUMAN; HUMAN CELL; NONHUMAN; OXIDATION; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT; SIGNAL TRANSDUCTION; THYMOCYTE;

ANIMALS; APOPTOSIS; GLUTATHIONE; HUMANS; NECROSIS; OXIDATIVE STRESS; T-LYMPHOCYTES;

EID: 4244006599     PISSN: 03784274     EISSN: None     Source Type: Journal    
DOI: 10.1016/0378-4274(95)03474-9     Document Type: Article

References (24)

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14. Dosedependent induction of apoptosis in human tumour cell lines by widely diverging stimuli
15. Different prooxidant levels stimulate cell growth, activate apoptosis, or produce necrosis in insulin-secreting RINmSF cells
16. Oxidized low density lipoproteins elicit DNA fragmentation of cultured lymphoblastoid cells
17. Lipid hydroperoxides induce apoptosis in T cells displaying a HIV-associated glutathione peroxidase deficiency
18. Thiol-mediated redox regulation of apoptosis
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21. Identification of a transitional preapoptotic population of thymocytes
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23. Apoptosis or necrosis intracellular levels of glutathione influence mode of cell death
24. Programmed cell death and bcl-2 protection in the absence of a nucleus