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Physiological and retinoid-induced proliferations of epidermis basal keratinocytes are differently controlled
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Retinoic acid is a high affinity selective ligand for the peroxisome proliferator-activated receptor
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Direct channeling of retinoic acid between cellular retinoic acid-binding protein II and retinoic acid receptor sensitizes mammary carcinoma cells to retinoic acid-induced growth arrest
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Selective cooperation between fatty acid binding proteins and peroxisome proliferators-activated receptors in regulating transcription
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Suppression of mammary carcinoma growth by retinoic acid: Proapoptotic genes are targets for retinoic acid receptor and cellular retinoic acid-binding protein II signaling
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The anti-apoptotic role of PPARbeta contributes to efficient skin wound healing
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