Lack of association of hepatic lipase polymorphisms with late-onset Alzheimer's disease

Neurobiology of Aging

Volumn 29, Issue 5, 2008, Pages 793-794

  Zhu, Haiyan ^a   Taylor, Jennie W ^a   Bennett, David A ^b   Younkin, Steven G ^c   Estus, Steven ^a  

^a UNIVERSITY OF KENTUCKY   (United States)

^b RUSH UNIVERSITY MEDICAL CENTER   (United States)

^c MAYO CLINIC   (United States)

Author keywords

Alzheimer's disease; Apolipoprotein E; Genetics; Hepatic lipase

Indexed keywords

APOLIPOPROTEIN E4; CHOLESTEROL; LIVER TRIACYLGLYCEROL LIPASE;

ALZHEIMER DISEASE; ARTICLE; GENE FREQUENCY; GENOTYPE; HUMAN; ONSET AGE; PRIORITY JOURNAL; SINGLE NUCLEOTIDE POLYMORPHISM;

AGED; AGED, 80 AND OVER; ALZHEIMER DISEASE; FEMALE; GENETIC PREDISPOSITION TO DISEASE; HUMANS; LIPASE; MALE; POLYMORPHISM, SINGLE NUCLEOTIDE; PREVALENCE; RISK ASSESSMENT; RISK FACTORS; STATISTICS AS TOPIC; UNITED STATES;

EID: 40849121018     PISSN: 01974580     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neurobiolaging.2006.11.015     Document Type: Article

References (2)

1. Baroni M.G., Berni A., Romeo S., Arca M., Tesorio T., Sorropago G., et al. Genetic study of common variants at the Apo E, Apo AI, Apo CIII, Apo B, lipoprotein lipase (LPL) and hepatic lipase (LIPC) genes and coronary artery disease (CAD): variation in LIPC gene associates with clinical outcomes in patients with established CAD. BMC Med. Genet. 4 (2003) 8
2. Knoblauch H., Bauerfeind A., Toliat M.R., Becker C., Luganskaja T., Gunther U.P., et al. Haplotypes and SNPs in 13 lipid-relevant genes explain most of the genetic variance in high-density lipoprotein and low-density lipoprotein cholesterol. Hum. Mol. Genet. 13 10 (2004) 993-1004