Activation of nicotinic acetylcholine receptor prevents the production of reactive oxygen species in fibrillar β amyloid peptide (1-42)-stimulated microglia

Experimental and Molecular Medicine

Volumn 40, Issue 1, 2008, Pages 11-18

  Ju, Hyun Moon ^a   Soo, Yoon Kim ^a   Hwan, Goo Lee ^a   Kim, Seung U ^a,b   Yong, Beom Lee ^a  

^a Ajou University School of Medicine   (South Korea)

^b UNIVERSITY OF BRITISH COLUMBIA   (Canada)

Author keywords

Acetylcholine; Adenosine triphosphate; Amyloid protein; Microglia; NADPH oxidase; Nicotine; Purinoceptor P2Z; Reactive oxygen species; Receptors, nicotinic

Indexed keywords

ADENOSINE TRIPHOSPHATE; ADENOSINE TRIPHOSPHATE DERIVATIVE; ALPHA BUNGAROTOXIN; AMYLOID BETA PROTEIN[1-42]; BUNGAROTOXIN RECEPTOR; CALCIUM; MECAMYLAMINE; NICOTINE; NICOTINIC RECEPTOR; PURINERGIC P2X7 RECEPTOR; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE;

ANIMAL CELL; ARTICLE; CALCIUM TRANSPORT; CELL STIMULATION; CONTROLLED STUDY; DRUG MECHANISM; ENZYME ACTIVATION; ENZYME INHIBITION; MICROGLIA; NONHUMAN; RAT; SIGNAL TRANSDUCTION;

EID: 40449129985     PISSN: 12263613     EISSN: 20926413     Source Type: Journal    
DOI: 10.3858/emm.2008.40.1.11     Document Type: Article

References (30)