Ulcerative colitis and adenocarcinoma of the colon in G alpha i2-deficient mice
Rudolph U, Finegold MJ, Rich SS, et al. Ulcerative colitis and adenocarcinoma of the colon in G alpha i2-deficient mice. Nature genetics 1995; 10:143-150.
TCR-mediated hyper-responsiveness of autoimmune Galphai2(-/-) mice is an intrinsic naive CD4(+) T cell disorder selective for the Galphai2 subunit
Huang TT, Zong Y, Dalwadi H, et al. TCR-mediated hyper-responsiveness of autoimmune Galphai2(-/-) mice is an intrinsic naive CD4(+) T cell disorder selective for the Galphai2 subunit. International immunology 2003; 15:1359-1367.
Regression of Peyer's patches in G alpha i2 deficient mice prior to colitis is associated with reduced expression of Bcl-2 and increased apoptosis
Ohman L, Franzen L, Rudolph U, Birnbaumer L, Hornquist EH. Regression of Peyer's patches in G alpha i2 deficient mice prior to colitis is associated with reduced expression of Bcl-2 and increased apoptosis. Gut 2002; 51:392-397.
Induction of colitis in mice deficient of Peyer's patches and mesenteric lymph nodes is associated with increased disease severity and formation of colonic lymphoid patches
Spahn TW, Herbst H, Rennert PD, et al. Induction of colitis in mice deficient of Peyer's patches and mesenteric lymph nodes is associated with increased disease severity and formation of colonic lymphoid patches. The American journal of pathology 2002; 161:2273-2282.
Transcriptional activation of the human Galphai2 gene promoter through nuclear factor-kappaB and antioxidant response elements
Arinze IJ, Kawai Y. Transcriptional activation of the human Galphai2 gene promoter through nuclear factor-kappaB and antioxidant response elements. J Biol Chem 2005; 280:9786-9795.