Sepsis: Redox mechanisms and therapeutic opportunities

Antioxidants and Redox Signaling

Volumn 9, Issue 11, 2007, Pages 1959-1961

  Biswal, Shyam ^a,c   Remick, Daniel G ^b  

^a The Johns Hopkins University   (United States)

^b BOSTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c JOHNS HOPKINS BLOOMBERG SCHOOL OF PUBLIC HEALTH   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CARBON MONOXIDE; MONOCYTE CHEMOTACTIC PROTEIN 1; NITRONE; OXYGEN RADICAL; PEROXYNITRITE; REACTIVE NITROGEN SPECIES; REACTIVE OXYGEN METABOLITE; SUPEROXIDE; TRANSCRIPTION FACTOR NRF2;

ARTIFICIAL VENTILATION; CAUSE OF DEATH; CELL FUNCTION; EDITORIAL; HUMAN; IMMUNOCOMPETENT CELL; LUNG INJURY; NITROSATIVE STRESS; OXIDATION REDUCTION REACTION; OXYGEN SUPPLY; PRIORITY JOURNAL; SEPSIS; TISSUE INJURY;

ANIMALS; CARBON MONOXIDE; HUMANS; OXIDATION-REDUCTION; REACTIVE OXYGEN SPECIES; RESPIRATION, ARTIFICIAL; SEPSIS;

EID: 35448970172     PISSN: 15230864     EISSN: None     Source Type: Journal    
DOI: 10.1089/ars.2007.1808     Document Type: Editorial

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