Tobacco smoke-induced left ventricular remodelling is not associated with metalloproteinase-2 or -9 activation

European Journal of Heart Failure

Volumn 9, Issue 11, 2007, Pages 1081-1085

  Castardeli, Edson ^a   Duarte, Daniella R ^a   Minicucci, Marcos F ^a   Azevedo, Paula S ^a   Matsubara, Beatriz B ^a   Matsubara, Luiz S ^a   Campana, Álvaro O ^a   Paiva, Sergio A R ^a   Zornoff, Leonardo A M ^a  

^a SÃO PAULO STATE UNIVERSITY UNESP   (Brazil)

Author keywords

Cardiac remodelling; Ventricular dilatation; Ventricular function

Indexed keywords

GELATINASE A; GELATINASE B; TOBACCO SMOKE;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DIASTOLE; ELECTROCARDIOGRAPHY MONITORING; ENZYME ACTIVATION; ENZYME ACTIVITY; ENZYME ASSAY; HEART LEFT VENTRICLE CONTRACTILITY; HEART LEFT VENTRICLE EJECTION FRACTION; HEART LEFT VENTRICLE FUNCTION; HEART MUSCLE CELL; HEART SIZE; HEART VENTRICLE REMODELING; MALE; MORPHOMETRICS; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; RAT; SMOKING; SYSTOLE; TISSUE SECTION;

ANIMALS; ECHOCARDIOGRAPHY; MALE; MATRIX METALLOPROTEINASE 2; MATRIX METALLOPROTEINASE 9; RATS; RATS, WISTAR; STATISTICS, NONPARAMETRIC; TOBACCO SMOKE POLLUTION; VENTRICULAR DYSFUNCTION, LEFT; VENTRICULAR REMODELING;

EID: 35448958879     PISSN: 13889842     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.ejheart.2007.09.004     Document Type: Article

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