Point:Counterpoint: Chronic hypoxia-induced pulmonary hypertension does/does not lead to loss of pulmonary vasculature [1]

Journal of Applied Physiology

Volumn 103, Issue 4, 2007, Pages 1455-

  Berg, John T ^a  

^a Virgen Milagrosa University Foundation   (Philippines)

Author keywords

[No Author keywords available]

Indexed keywords

FUNCTIONAL MAGNETIC RESONANCE IMAGING; HUMAN; HYPOXIA; LETTER; LUNG EDEMA; LUNG VASOCONSTRICTION; NONHUMAN; PRIORITY JOURNAL; PULMONARY ARTERY; PULMONARY HYPERTENSION; ANIMAL; ANOXIA; ARTIFACT; CHRONIC DISEASE; DISEASE MODEL; LUNG; LUNG CIRCULATION; METABOLISM; NEOVASCULARIZATION (PATHOLOGY); NOTE; NUCLEAR MAGNETIC RESONANCE IMAGING; PATHOLOGY; PATHOPHYSIOLOGY; PERFUSION; PHYSIOLOGY; VASCULAR ENDOTHELIUM; VASCULAR SMOOTH MUSCLE; VASCULARIZATION; VASOCONSTRICTION;

ANIMALS; ANOXIA; ARTIFACTS; CHRONIC DISEASE; DISEASE MODELS, ANIMAL; ENDOTHELIUM, VASCULAR; HUMANS; HYPERTENSION, PULMONARY; LUNG; MAGNETIC RESONANCE IMAGING; MUSCLE, SMOOTH, VASCULAR; NEOVASCULARIZATION, PATHOLOGIC; PERFUSION; PULMONARY ARTERY; PULMONARY CIRCULATION; VASOCONSTRICTION;

EID: 35348961449     PISSN: 87507587     EISSN: 15221601     Source Type: Journal    
DOI: 10.1152/japplphysiol.00739.2007     Document Type: Letter

References (6)

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2. deMello D, Reid LM. Arteries and veins. In: The Lung: Scientific Foundations, edited by Crystal RG and West JB. New York: Raven, 1991, p. 767-777.
3. Hopkins SR, Garg J, Bolar DS, Balouch J, Levin DL. Pulmonary blood flow heterogeneity during hypoxia and high-altitude pulmonary edema. Am J Respir Crit Care Med 171: 83-87, 2005.
4. Hultgren HN. High altitude pulmonary edema. In: Biomedicine of High Terrestrial Altitude, edited by Hegnauer AH. New York: Springer-Verlag, 1969, p. 131-141.
5. McLoughlin P, McMurtry I. Counterpoint: Chronic hypoxia-induced pulmonary hypertension does not lead to loss of pulmonary vasculature. J Appl Physiol; doi:10.1152/japplphysiol.00274a.2007.
6. Rabinovitch M, Chesler N, Molthen RC. Point: Chronic hypoxia-induced pulmonary hypertension does lead to loss of pulmonary vasculature. J Appl Physiol; doi:10.1152/japplphysiol.00274.2007.