By blocking apoptosis, Bcl-2 in p38-dependent manner promotes cell cycle arrest and accelerated senescence after DNA damage and serum withdrawal

Cell Cycle

Volumn 6, Issue 17, 2007, Pages 2171-2177

  Nelyudova, Anna ^a   Aksenov, Nikolay ^a   Pospelov, Valery ^a   Pospelova, Tatiana ^a  

^a INSTITUTE OF CYTOLOGY   (Russian Federation)

Author keywords

Adriamycin; Bcl 2; Cell cycle arrest; Cell senescence; E1A; p38 MAPK; Ras

Indexed keywords

DOXORUBICIN; E1A PROTEIN; MITOGEN ACTIVATED PROTEIN KINASE P38; NOCODAZOLE; PROTEIN BCL 2; RAS PROTEIN;

APOPTOSIS; ARTICLE; CELL AGING; CELL CYCLE ARREST; CONTROLLED STUDY; DNA DAMAGE; GENE DELETION; HUMAN; HUMAN CELL; PROTEIN EXPRESSION;

RODENTIA;

EID: 34548824056     PISSN: 15384101     EISSN: 15514005     Source Type: Journal    
DOI: 10.4161/cc.6.17.4610     Document Type: Article

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