Parvalbumin isoforms differentially accelerate cardiac myocyte relaxation kinetics in an animal model of diastolic dysfunction

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 293, Issue 3, 2007, Pages

  Rodenbaugh, David W ^a   Wang, Wang ^a   Davis, Jennifer ^a   Edwards, Terri ^a   Potter, James D ^b   Metzger, Joseph M ^a  

^a UNIVERSITY OF MICHIGAN MEDICAL SCHOOL   (United States)

^b UNIVERSITY OF MIAMI MILLER SCHOOL OF MEDICINE   (United States)

Author keywords

Gene transfer; Mechanical relaxation

Indexed keywords

ADENOSINE TRIPHOSPHATASE (POTASSIUM SODIUM); ADENOVIRUS VECTOR; ALPHA PARVALBUMIN; BETA PARVALBUMIN; PARVALBUMIN; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BODY WEIGHT; CONGESTIVE HEART FAILURE; CONTROLLED STUDY; DIASTOLIC DYSFUNCTION; ECHOCARDIOGRAPHY; GENE TRANSFER; HEART HEMODYNAMICS; HEART MUSCLE CELL; HEART MUSCLE RELAXATION; HYPERTENSION; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; RAT; SALT INTAKE;

ADENOVIRIDAE; ANIMALS; CELLS, CULTURED; DISEASE MODELS, ANIMAL; DISEASE PROGRESSION; ECHOCARDIOGRAPHY; GENE TRANSFER TECHNIQUES; HEART FAILURE, CONGESTIVE; HYPERTENSION; KINETICS; MALE; MYOCARDIAL CONTRACTION; MYOCARDIUM; MYOCYTES, CARDIAC; PARVALBUMINS; PROTEIN ISOFORMS; RATS; RATS, INBRED DAHL; SARCOMERES;

EID: 34548436142     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00232.2007     Document Type: Article

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