Misfolded transthyretin causes behavioral changes in a Drosophila model for transthyretin-associated amyloidosis

European Journal of Neuroscience

Volumn 26, Issue 4, 2007, Pages 913-924

  Pokrzywa, Malgorzata ^a   Dacklin, Ingrid ^a   Hultmark, Dan ^a   Lundgren, Erik ^a  

^a UMEÅ UNIVERSITY   (Sweden)

Author keywords

Amyloid; Amyloidosis; Drosophila; Familial amyloidotic polyneuropathy; Neurodegeneration; Transthyretin

Indexed keywords

AMYLOID; MUTANT PROTEIN; PREALBUMIN;

ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ATTENUATION; BEHAVIOR CHANGE; BODY POSTURE; DISEASE MODEL; DROSOPHILA; FAMILIAL AMYLOID POLYNEUROPATHY; FEMALE; FLY; FLYING; FORELIMB; GENE; LIFESPAN; LOCOMOTION; LONGEVITY; MALE; MOLECULE; NERVE DEGENERATION; NONHUMAN; PRIORITY JOURNAL; PROTEIN AGGREGATION; PROTEIN ENGINEERING; PROTEIN EXPRESSION; PROTEIN VARIANT; SCREENING; TIME; TRANSGENIC ANIMAL; WILD TYPE;

AGING; AMYLOIDOSIS; ANIMALS; BEHAVIOR, ANIMAL; BLOTTING, WESTERN; DNA, COMPLEMENTARY; DROSOPHILA MELANOGASTER; FLIGHT, ANIMAL; HEMOLYMPH; IMMUNOHISTOCHEMISTRY; LONGEVITY; MICROSCOPY, CONFOCAL; MOTOR ACTIVITY; NEUROPEPTIDES; PHENOTYPE; PREALBUMIN; PROTEIN FOLDING; TRANSGENES; WING;

EID: 34547969339     PISSN: 0953816X     EISSN: 14609568     Source Type: Journal    
DOI: 10.1111/j.1460-9568.2007.05728.x     Document Type: Article

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