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Volumn 157, Issue 3, 2007, Pages 523-530

p16ink4a expression decreases during imiquimod treatment of anal intraepithelial neoplasia in human immunodeficiency virus-infected men and correlates with the decline of lesional high-risk human papillomavirus DNA load

Author keywords

Anal intraepithelial neoplasia; Human immunodeficiency virus infection; Human papillomavirus DNA load; Human papillomavirus infection; p16ink4a; Proliferative biomarkers

Indexed keywords

CYCLIN DEPENDENT KINASE INHIBITOR 1; IMIQUIMOD; KI 67 ANTIGEN; MINICHROMOSOME MAINTENANCE PROTEIN; PROTEIN P16INK4A; VIRUS DNA; AMINOQUINOLINE DERIVATIVE; ANTINEOPLASTIC AGENT; CYCLIN DEPENDENT KINASE INHIBITOR 2A; TUMOR MARKER;

EID: 34547925605     PISSN: 00070963     EISSN: 13652133     Source Type: Journal    
DOI: 10.1111/j.1365-2133.2007.08004.x     Document Type: Article
Times cited : (29)

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