Why type 2 diabetes is not an equal opportunity disease: Recent progress in genetics

Current Opinion in Clinical Nutrition and Metabolic Care

Volumn 10, Issue 4, 2007, Pages 389-390

  Doria, Alessandro ^a   Abumrad, Nada A ^b,c  

^a JOSLIN DIABETES CENTER   (United States)

^b WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c WASHINGTON UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

HEPATOCYTE NUCLEAR FACTOR 4ALPHA; MEMBRANE PROTEIN; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA; TRANSCRIPTION FACTOR 7;

DISEASE PREDISPOSITION; EDITORIAL; GENETIC HETEROGENEITY; GENETIC POLYMORPHISM; GENETICS; HEREDITY; HIGH RISK POPULATION; INSULIN SENSITIVITY; LIPID METABOLISM; MATURITY ONSET DIABETES MELLITUS; NON INSULIN DEPENDENT DIABETES MELLITUS; RACE;

EID: 34250613427     PISSN: 13631950     EISSN: 15353885     Source Type: Journal    
DOI: 10.1097/MCO.0b013e32821c6174     Document Type: Editorial

References (3)

1. Mokdad AH, Bowman BA, Ford ES, et al. The continuing epidemics of obesity and diabetes in the United States. JAMA 2001; 286:1195-1200.
2. th ed New York: Lippincott Williams & Wilkins; 2005.
3. Sladek R, Rocheleau G, Rung J, et al. A genome-wide association study identifies novel risk loci for type 2 diabetes. Nature 2007; 445:881-885.