Redox imbalance in diabetes

Antioxidants and Redox Signaling

Volumn 9, Issue 7, 2007, Pages 865-867

  Hayden, Melvin R ^a,b   Sowers, James R ^a  

^a University of Missouri   (United States)

^b Health Sciences Center Division of Nephrology Department of Internal Medicine   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GLUCOSE; HOMOCYSTEINE; INSULIN; NICOTINAMIDE ADENINE DINUCLEOTIDE; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA AGONIST; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; CARDIOVASCULAR DISEASE; DIABETES MELLITUS; EDITORIAL; GLUCOSE BLOOD LEVEL; HUMAN; HYPERINSULINEMIA; IMPAIRED GLUCOSE TOLERANCE; INSULIN DEPENDENT DIABETES MELLITUS; INSULIN RESISTANCE; MOUSE; NON INSULIN DEPENDENT DIABETES MELLITUS; NONHUMAN; OBESITY; OXIDATION REDUCTION REACTION; PANCREAS ISLET BETA CELL; PRIORITY JOURNAL; RENIN ANGIOTENSIN ALDOSTERONE SYSTEM;

ANIMALS; DIABETES MELLITUS; DIABETES MELLITUS, TYPE 2; HUMANS; OXIDATION-REDUCTION; OXIDATIVE STRESS;

EID: 34250338918     PISSN: 15230864     EISSN: None     Source Type: Journal    
DOI: 10.1089/ars.2007.1640     Document Type: Editorial

References (14)

1. Amos AF, McCarty DJ, and Zimmet P. The rising global burden of diabetes and its complications: estimates and projections to the year 2010. Diabetes Med 14: S1-S85, 1997.
2. Arslanian SA. Type 2 diabetes mellitus in children: pathophysiology and risk factors. J Pediatr Endocrinol Metab 13(suppl 6): 1385-1394, 2000.
3. Chu KY and Leung PS. Angiotensin II type I receptor antagonism mediates uncoupling protein 2-driven oxidative stress and ameliorates pancreatic islet β-cell function in young type 2 diabetic mice. Antioxid Redox Signal 9: 869-878, 2007.
4. Nicolls MR, Haskins K, and Flores SC. Oxidant stress, immune dysregulation, and vascular function in type I diabetes. Antioxid Redox Signal 9: 879-889, 2007.
5. Hayden MR. Islet amyloid, metabolic syndrome and the natural progressive history of type 2 diabetes mellitus. JOP.J Pancreas (online) 3: 126-138, 2002.
6. Hayden MR and Sowers JR. Isletopathy in type 2 diabetes mellitus: implications of islet RAS, islet fibrosis, islet amyloid, remodeling, and oxidative stress. Antioxid Redox Signal 9: 891-910, 2007.
7. Hayden MR, Poorna R, Karuparthi PR, Manrique CM, Lastra G, Habibi J, and Sowers JR. Longitudinal ultrastructure study of islet amyloid in the HIP rat model of type 2 diabetes mellitus. Exp Biol Med (in press).
8. Hayden MR, Stump C, and Sowers JR. Organ involvement in cardiometabolic syndrome. J Cardiometab Syndr 1: 16-24, 2006.
9. Ido Y. Pyridine nucleotide redox abnormalities in diabetes. Antioxid Redox Signal 9: 931-942, 2007.
10. Lastra G and Manrique MC. The expanding role of oxidative stress, renin angiotensin system, and β-cell dysfunction in the cardiometabolic syndrome and type 2 diabetes mellitus. Antioxid Redox Signal 9: 943-954, 2007.
11. Pennathur S and Heinecke JW. Mechanisms for oxidative stress in diabetic cardiovascular disease. Antioxid Redox Signal 9: 955-969, 2007.
12. Sen U, Tyagi N, Moshal KS, Kartha GK, Rosenberger D, Henderson BC, Joshua IG, and Tyagi SC. Cardiac synchronous and dys-synchronous remodeling in diabetes mellitus. Antioxid Redox Signal 9: 971-978, 2007.
13. Shah S, Iqbal M, Karam J, Salifu M, and McFarlane SI. Oxidative stress, glucose metabolism, and the prevention of type 2 diabetes: pathophysiological insights. Antioxid Redox Signal 9: 911-929, 2007.
14. Zimmel O. The burden of type 2 diabetes: are we doing enough? Diabetes Metab 29: 6S9-6S18, 2003.