Induction of high STAT1 expression in transgenic mice with LQTS and heart failure

Biochemical and Biophysical Research Communications

Volumn 358, Issue 2, 2007, Pages 449-454

  Wu, Ling ^a,c   Archacki, Stephen R ^a,c   Zhang, Teng ^a,b   Wang, Qing K ^a,b,c,d  

^a LERNER RESEARCH INSTITUTE   (United States)

^b CASE WESTERN RESERVE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c CLEVELAND STATE UNIVERSITY   (United States)

^d HUAZHONG UNIVERSITY OF SCIENCE AND TECHNOLOGY   (China)

Author keywords

Cardiac sodium channel gene SCN5A mutation N1325S; Dilated cardiomyopathy and heart failure; Long QT syndrome (LQTS) and ventricular tachycardia; Microarray analysis; STAT1

Indexed keywords

STAT1 PROTEIN;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DNA MICROARRAY; GENE IDENTIFICATION; GENETIC VARIABILITY; HEART FAILURE; LONG QT SYNDROME; MALE; MICROARRAY ANALYSIS; MOUSE; NONHUMAN; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN INDUCTION; REAL TIME POLYMERASE CHAIN REACTION; TRANSGENIC MOUSE; WESTERN BLOTTING;

ANIMALS; CARDIAC OUTPUT, LOW; LONG QT SYNDROME; MICE; MICE, KNOCKOUT; MICE, TRANSGENIC; MYOCARDIUM; PROTEOME; SIGNAL TRANSDUCTION; SODIUM CHANNELS; STAT1 TRANSCRIPTION FACTOR; UP-REGULATION;

MUS; MUS MUSCULUS;

EID: 34249009224     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2007.04.119     Document Type: Article

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