Nitric oxide-dependent and -independent mechanisms are involved in TNF-α-induced depression of cardiac myocyte contractility

American Journal of Physiology - Regulatory Integrative and Comparative Physiology

Volumn 292, Issue 5, 2007, Pages

  Kumar, Anand ^a,b,d   Paladugu, Bhanu ^a   Mensing, Joel ^a   Kumar, Aseem ^c   Parrillo, Joseph E ^a  

^a RUTGERS ROBERT WOOD JOHNSON MEDICAL SCHOOL   (United States)

^b UNIVERSITY OF MANITOBA   (Canada)

^c LAURENTIAN UNIVERSITY   (Canada)

^d HEALTH SCIENCES CENTRE   (Canada)

Author keywords

Adrenoreceptor; Cytokine; Heart; Myocardial depressant substance; Myocardial depression; Myocyte; Sepsis; Septic shock

Indexed keywords

BUCLADESINE; CALCIUM CHLORIDE; CYCLIC AMP; FORSKOLIN; ISOPRENALINE; NITRIC OXIDE; NITRIC OXIDE SYNTHASE INHIBITOR; TUMOR NECROSIS FACTOR ALPHA;

ADRENERGIC ACTIVITY; ANIMAL CELL; ANIMAL TISSUE; ARTICLE; CALCIUM SIGNALING; CONTROLLED STUDY; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILITY; HEART MUSCLE EXCITABILITY; HEART MUSCLE RELAXATION; IN VITRO STUDY; NONHUMAN; PRIORITY JOURNAL; RAT; SIGNAL TRANSDUCTION; TISSUE CULTURE;

ANIMALS; ARGININE; CELLS, CULTURED; CYCLIC AMP; DOSE-RESPONSE RELATIONSHIP, DRUG; FORSKOLIN; ISOPROTERENOL; MYOCARDIAL CONTRACTION; MYOCYTES, CARDIAC; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; RATS; RATS, SPRAGUE-DAWLEY; TUMOR NECROSIS FACTOR-ALPHA;

EID: 34248209553     PISSN: 03636119     EISSN: 15221490     Source Type: Journal    
DOI: 10.1152/ajpregu.00146.2006     Document Type: Article

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