Induced increase in virulence of low pathogenic avian influenza by serial intracerebral passage in chickens

Avian Diseases

Volumn 51, Issue SUPPL. 1, 2007, Pages 396-400

  Londt B Z ^a   Banks, J ^a   Gardner, R ^a   Cox, W J ^a   Brown, I H ^a  

^a VETERINARY LABORATORIES AGENCY   (United Kingdom)

Author keywords

Avian influenza; Hemagglutinin; ICPI; Pathogenicity; Poultry; Serial

Indexed keywords

AVES; AVIAN INFLUENZA VIRUS; GALLUS GALLUS; ORTHOMYXOVIRIDAE;

EID: 34247581910     PISSN: 00052086     EISSN: None     Source Type: Journal    
DOI: 10.1637/7665-061206r.1     Document Type: Conference Paper

References (5)

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2. Alexander, D. J., S. A. Lister, M. J. Johnson, C. J. Randall, and P. J. Thomas. An outbreak of highly pathogenic avian influenza in turkeys in Great Britain in 1991. Vet. Rec. 132:535-536. 1993.
3. Banks, J., and L. Plowright. Additional glycosylation at the receptor binding site of hemagglutinin (HA) for H5 and H7 viruses may be an adaptation to poultry hosts, but does it influence pathogenicity. Avian Dis. 47:942-950. 2003.
4. OIE (World Organisation for Animal Health). OIE manual of diagnostic tests and vaccines for terrestrial animals (mammals, birds and bees), chap 2.1.14, highly pathogenic avian influenza, vol 1, 5th ed. 2004. http://www.oie.int/eng/normes/mmanual/A_summry.htm.
5. Wood, G. W., J. Banks, J. W. McCauley, and D. J. Alexander. Deduced amino acid sequences of the hemagglutinin of H5N1 avian influenza virus isolates from an outbreak in turkeys in Norfolk, England. Arch. Virol. 134:185-194. 1994.