Sudden infant death syndrome and brain abnormalities [1]

JAMA

Volumn 297, Issue 11, 2007, Pages 1190-

  Guntheroth, Warren G ^a   Spiers, Philip S ^a  

^a UNIVERSITY OF WASHINGTON SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

NEUROTRANSMITTER; SEROTONIN; SEROTONIN RECEPTOR;

ANEMIA; ASPHYXIA; BRAIN DISEASE; BRAIN STEM; CEREBROSPINAL FLUID; DISEASE COURSE; HUMAN; HYPOXIA; LETTER; MORTALITY; PRIORITY JOURNAL; PROTEIN FUNCTION; RISK ASSESSMENT; SEROTONINERGIC SYSTEM; SUDDEN INFANT DEATH SYNDROME; BRAIN; INFANT; NEWBORN; NOTE; PATHOLOGY; CAUSE OF DEATH; TOXICITY; FETUS HYPOXIA; GLIOSIS; METABOLISM;

BRAIN; HUMANS; INFANT; INFANT, NEWBORN; SUDDEN INFANT DEATH; FETAL HYPOXIA; GLIOSIS; RECEPTORS, SEROTONIN;

EID: 33947546078     PISSN: 00987484     EISSN: 15383598     Source Type: Journal    
DOI: 10.1001/jama.297.11.1190-a     Document Type: Letter

References (5)

1. Paterson DS, Trachtenberg FL, Thompson EG, et al. Multiple serotonergic brainstem abnormalities in sudden infant death syndrome. JAMA. 2006;296:2124-2132.
2. Waggener TB, Southall DP, Scott LA. Analysis of breathing patterns in a prospective population of term infants does not predict susceptibility to SIDS. Pediatr Res. 1990;27:113-117.
3. Guntheroth WG, Spiers PS. The triple risk hypothesis in sudden infant death syndrome. Pediatrics. 2002;110:e64.
4. Cann-Moisan C, Girin E, Giroux JD, Le Bras P, Caroff J. Changes in cerebrospinal fluid monomine metabolites, tryptophan, and gamma-aminobutyric acid during the 1st year of life in normal infants: comparison with victims of sudden infant death syndrome. Biol Neonate. 1999;75:152-159.
5. Del Bigio MR, Becker LE. Microglial aggregation in the dentate gyrus: a marker of mild hypoxic-ischemic brain insult in human infants. Neuropathol Appl Neurobiol. 1994;20:144-151.