Reply to STOX1 is not imprinted and is not likely to be involved in preeclampsia [2]

Nature Genetics

Volumn 39, Issue 3, 2007, Pages 280-281

  Van Dijk, Marie ^a   Van Bezu, Jan ^a   Chim, Stephen S ^b   Lo, Y M Dennis ^b   Blankenstein, Marinus A ^a   Oudejans, Cees B M ^a  

^a VU UNIVERSITY MEDICAL CENTER   (Netherlands)

^b CHINESE UNIVERSITY OF HONG KONG   (Hong Kong)

Author keywords

[No Author keywords available]

Indexed keywords

CYCLIN DEPENDENT KINASE INHIBITOR 1C;

ALLELE; CELL TYPE; CPG ISLAND; CTNNA3 GENE; DISEASE ASSOCIATION; EPIGENETICS; EXPERIMENTAL ANIMAL; EXPERIMENTAL MODEL; GENE; GENE INTERACTION; GENETIC ASSOCIATION; GENETIC POLYMORPHISM; GENETIC RISK; GENETIC SUSCEPTIBILITY; GENETIC VARIABILITY; HUMAN; INTRAUTERINE GROWTH RETARDATION; LETTER; NONHUMAN; PHENOTYPE; PREECLAMPSIA; PRIORITY JOURNAL; SEX RATIO; STOX1 GENE; TRANSACTIVATION;

EID: 33847317325     PISSN: 10614036     EISSN: 15461718     Source Type: Journal    
DOI: 10.1038/ng0307-280     Document Type: Letter

References (7)

1. Iglesias-Platas, I. et al. Nat. Genet. 39, 279-280 (2007).
2. Van Dijk, M. et al. Nat. Genet. 37, 514-519 (2005).
3. Oudejans, C.B. et al. Hum. Genet. 120, 607-612 (2007).
4. Oudejans, C.B. et al. Mol. Hum. Reprod. 10, 589-598 (2004).
5. Van Dijk, M. et al. Gene Expr. Patterns 4, 61-65 (2004).
6. Saleem, R.A. et al. Hum. Mol. Genet. 12, 2993-3005 (2003).
7. Lo, Y.M.D. et al. Nat. Med. 13, 218-223 (2007).