Herpes Simplex Virus type 2 (HSV-2) establishes latent infection in a different population of ganglionic neurons than HSV-1: Role of latency-associated transcripts

Journal of Virology

Volumn 81, Issue 4, 2007, Pages 1872-1878

  Margolis, Todd P ^a,b   Imai, Yumi ^a   Yang, Li ^a,d   Vallas, Vicky ^a,e   Krause, Philip R ^c  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

^c CENTER FOR BIOLOGICS EVALUATION AND RESEARCH   (United States)

^d AMGEN INC   (United States)

^e AUSTIN RESEARCH INSTITUTE   (Australia)

Author keywords

[No Author keywords available]

Indexed keywords

LATENCY ASSOCIATED PEPTIDE;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CHIMERA; CONTROLLED STUDY; FEMALE; HERPES SIMPLEX VIRUS 1; HERPES SIMPLEX VIRUS 2; LATENT VIRUS INFECTION; MOUSE; NERVE CELL; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; SENSORY GANGLION; SPINAL GANGLION; TRIGEMINAL NERVE; VIRUS MUTANT;

ANIMALS; GANGLIA; HERPES SIMPLEX; HERPESVIRUS 1, HUMAN; HERPESVIRUS 2, HUMAN; MICE; NEURONS; SPECIES SPECIFICITY; VIRULENCE; VIRUS LATENCY;

HUMAN HERPESVIRUS 1; HUMAN HERPESVIRUS 2; MURINAE; SIMPLEXVIRUS;

EID: 33846787137     PISSN: 0022538X     EISSN: None     Source Type: Journal    
DOI: 10.1128/JVI.02110-06     Document Type: Article

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