Osteonecrosis of the jaw - A potential adverse effect of bisphosphonate treatment: Commentary

Nature Clinical Practice Endocrinology and Metabolism

Volumn 2, Issue 12, 2006, Pages 662-663

  Felsenberg, Dieter ^a  

^a HUMBOLDT UNIVERSITY   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

BISPHOSPHONIC ACID DERIVATIVE; PAMIDRONIC ACID; ZOLEDRONIC ACID;

BONE NECROSIS; BREAST CANCER; CLINICAL PRACTICE; DENTAL CARE; DENTAL SURGERY; DRUG MEGADOSE; DRUG POTENCY; HUMAN; JAW DISEASE; METASTASIS; MOUTH INFECTION; MULTIPLE MYELOMA; NOTE; OSTEOPOROSIS; PAGET BONE DISEASE; PRACTICE GUIDELINE; PRIORITY JOURNAL; RISK FACTOR; SYSTEMATIC REVIEW; TREATMENT OUTCOME;

EID: 33845459836     PISSN: 17458366     EISSN: 17458374     Source Type: Journal    
DOI: 10.1038/ncpendmet0348     Document Type: Note

References (4)

1. Mashiba T et al. (2005) The effects of suppressed bone remodeling by bisphosphonates on microdamage accumulation and degree of mineralization in the cortical bone of dog rib. J Bone Miner Metab 23 (Suppl): 36-42
2. Komatsubara S et al. (2004) Suppressed bone turnover by long-term bisphosphonate treatment accumulates microdamage but maintains intrinsic material properties in cortical bone of dog rib. J Bone Miner Res 19: 999-1005
3. Komatsubara S et al. (2003) Long-term treatment of incadronate disodium accumulates microdamage but improves the trabecular bone microarchitecture in dog vertebra. J Bone Miner Res 18: 512-520
4. Burr DB et al. (2003) Tissue mineralization is increased following 1-year treatment with high doses of bisphosphonates in dogs. Bone 33: 960-969