Intrauterine growth after uterine artery ligation in rats: Dependence on the fetal position in the uterine horn and need for prenatal marking of the animals [1]

Journal of Hypertension

Volumn 25, Issue 1, 2007, Pages 247-248

  Nüsken, Kai Dietrich ^a   Warnecke, Christina ^a   Hilgers, Karl F ^a   Schneider, Holm ^a  

^a UNIVERSITY OF ERLANGEN NUREMBERG   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

SOMATOMEDIN BINDING PROTEIN 1;

AGED; AGING; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTERY COMPLIANCE; ARTERY LIGATION; CONTROLLED STUDY; FETUS WEIGHT; HYPERTENSION; INTRAUTERINE GROWTH RETARDATION; LETTER; NONHUMAN; PLACENTA CIRCULATION; PLACENTA INSUFFICIENCY; PRENATAL GROWTH; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; RIGIDITY; STRESS; UTERINE ARTERY;

EID: 33845361769     PISSN: 02636352     EISSN: None     Source Type: Journal    
DOI: 10.1097/01.hjh.0000254371.70823.d4     Document Type: Letter

References (5)

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2. Denton KM. Can adult cardiovascular disease be programmed in utero? J Hypertens 2006; 24:1245-1247.
3. Wigglesworth J. Experimental growth retardation in the foetal rat. J Pathol Bacteriol 1964; 88:1-13.
4. Watson CS, Bialek P, Anzo M, Khosravi J, Jee SP, Han VK. Elevated circulating insulin-like growth factor binding protein-1 is sufficient to cause fetal growth restriction. Endocrinology 2006; 147:1175-1186.
5. Waddington SN, Buckley SMK, Nivsarkar M, Jezzard S, Schneider H, Dahse T, et al. In utero gene transfer of human factor IX to fetal mice can induce postnatal tolerance of the exogenous clotting factor. Blood 2003; 101:1359-1366.