In atherogenesis, the apoptosis of endothelial cell itself could directly induce over-proliferation of smooth muscle cells

Medical Hypotheses

Volumn 68, Issue 2, 2007, Pages 275-277

  Qin, Chunchang ^a   Liu, Zhihong ^a  

^a FUWAI HOSPITAL   (China)

Author keywords

[No Author keywords available]

Indexed keywords

DIPEPTIDYL CARBOXYPEPTIDASE INHIBITOR; HYDROXYMETHYLGLUTARYL COENZYME A REDUCTASE INHIBITOR; NITRIC OXIDE; PROSTAGLANDIN;

APOPTOSIS; ARTICLE; ATHEROGENESIS; CELL ACTIVITY; CELL DEATH; CELL DIFFERENTIATION; CELL FUNCTION; CELL PROLIFERATION; ENDOTHELIAL DYSFUNCTION; ENDOTHELIUM CELL; ENERGY CONSUMPTION; HUMAN; NONHUMAN; PRIORITY JOURNAL; SMOOTH MUSCLE FIBER;

APOPTOSIS; ATHEROSCLEROSIS; CELL DIVISION; ENDOTHELIUM, VASCULAR; HUMANS; HYDROXYMETHYLGLUTARYL-COA REDUCTASE INHIBITORS; MUSCLE, SMOOTH, VASCULAR;

EID: 33845344987     PISSN: 03069877     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.mehy.2006.07.037     Document Type: Article

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