Mechanism of cerebellar function studied using mutant mice [5]

Cerebellum

Volumn 5, Issue 4, 2006, Pages 299-300

  Hirano, Tomoo ^a,b   Takashi, Yoshida ^a,b   Funabiki, Kazuo ^a,b  

^a KYOTO UNIVERSITY   (Japan)

^b JAPAN SCIENCE AND TECHNOLOGY AGENCY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

GLUTAMATE RECEPTOR; GLUTAMATE RECEPTOR DELTA2;

BRAIN FUNCTION; CEREBELLUM; CLIMBING FIBER; LETTER; MOTOR CONTROL; MOTOR DYSFUNCTION; MOUSE; MOUSE MUTANT; NEUROPHYSIOLOGY; NONHUMAN; OPTOKINETIC STIMULATION; PRIORITY JOURNAL; PURKINJE CELL; SYNAPSE; VESTIBULOOCULAR REFLEX; BRAIN REGION; JAPAN;

ANIMALS; ASSOCIATION LEARNING; CEREBELLUM; HUMANS; LEARNING; MEMORY; MICE; MICE, MUTANT STRAINS; MOTOR ACTIVITY; NEURONS; SAIMIRI;

EID: 33845312971     PISSN: 14734222     EISSN: 14734230     Source Type: Journal    
DOI: 10.1080/14734220600776387     Document Type: Letter

References (3)

1. Kashiwabuchi N, Ikeda K, Araki K, Hirano T, Shibuki K, Takayama C, et al. Disturbed motor coordination, Purkinje cell synapse formation and cerebellar long-term depression of mice defective in the δ2 subunit of the glutamate receptor channel. Cell. 1995;81:245-52.
2. Yoshida T, Katoh A, Ohtsuki G, Mishina M, Hirano T. Oscillating Purkinje neuron activity causing involuntary eye movement in a mutant mouse deficient in the glutamate receptor δ2 subunit. J Neurosci. 2004;24:2440-8.
3. Katoh A, Yoshida T, Himeshima Y, Mishina M, Hirano T. Defective control and adaptation of reflex eye movements in mutant mice deficient in either the glutamate receptor δ2 subunit or Purkinje cells. Eur J Neurosci. 2005;21:1315-26.