Stability of genetically engineered cardiac pacemaker - Role of intracellular Ca2+ handling

Computers in Cardiology

Volumn 32, Issue , 2005, Pages 969-972

  Zhang, H ^a   Tong, W C ^b   Garratt, C J ^a   Holden, A V ^b  

^a UNIVERSITY OF MANCHESTER   (United Kingdom)

^b UNIVERSITY OF LEEDS   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM; CARDIOVASCULAR SYSTEM; COMPUTER SIMULATION; GENETIC ENGINEERING; MUSCLE; SYSTEM STABILITY;

MYOCYTES; POTASSIUM CURRENT; VENTRICULAR MUSCLE;

PACEMAKERS;

EID: 33750455707     PISSN: 02766574     EISSN: None     Source Type: Conference Proceeding    
DOI: 10.1109/CIC.2005.1588270     Document Type: Conference Paper

References (7)

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3. 2+ content in isolated failing and non-failing human myocardium. Circulation Research 1999; 85: 38-46.
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6. Lee KS, Marban E, Tsien RW. Inactivation of calcium channels in mammalian heart cells: joint dependence on membrane potential and intracellular calcium. Journal of Physiology 1985; 364: 395-411.
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