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A mechanism converting psychosocial stress into mononuclear cell activation
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The role of IL-1beta in stress-induced sensitization of proinflammatory cytokine and corticosterone responses
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When not enough is too much: The role of insufficient glucocorticoid signaling in the pathophysiology of stress-related disorders
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Raison CL, Miller AH: When not enough is too much: the role of insufficient glucocorticoid signaling in the pathophysiology of stress-related disorders. Am J Psychiatry 2003; 160:1554-1565
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