Spinal cord injury-specific depression of monosynaptic spinal reflex transmission by l-5-hydroxytryptophan results from loss of the 5-HT uptake system and not 5-HT receptor supersensitivity

Experimental Neurology

Volumn 202, Issue 1, 2006, Pages 258-261

  Honda, Motoko ^a   Tanabe, Mitsuo ^a   Ono, Hideki ^a  

^a NAGOYA CITY UNIVERSITY   (Japan)

Author keywords

5 HT; 5 HT reuptake; Spinal cord injury; Spinal reflexes

Indexed keywords

5 HYDROXYTRYPTOPHAN; SEROTONIN 1B AGONIST; SEROTONIN 1D AGONIST; SEROTONIN RECEPTOR; SEROTONIN TRANSPORTER; SUMATRIPTAN SUCCINATE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; DORSAL ROOT; FEMALE; MONOSYNAPTIC REFLEX; NERVE STIMULATION; NONHUMAN; POSTSYNAPTIC POTENTIAL; PRESYNAPTIC MEMBRANE; PRIORITY JOURNAL; RAT; SEROTONIN UPTAKE; SEROTONINERGIC TRANSMISSION; SPINAL CORD INJURY; SPINAL REFLEX; SUPERSENSITIVITY; SYNAPTIC TRANSMISSION; VENTRAL ROOT;

5-HYDROXYTRYPTOPHAN; ANALYSIS OF VARIANCE; ANIMALS; ANTIDEPRESSIVE AGENTS, SECOND-GENERATION; DISEASE MODELS, ANIMAL; FEMALE; RATS; RATS, WISTAR; RECEPTORS, SEROTONIN; REFLEX, MONOSYNAPTIC; SEROTONIN; SEROTONIN AGENTS; SPINAL CORD INJURIES; TIME FACTORS;

EID: 33748418432     PISSN: 00144886     EISSN: 10902430     Source Type: Journal    
DOI: 10.1016/j.expneurol.2006.05.022     Document Type: Article

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