Hypoxia: A common mechanism of tumorigenesis?

Medical Hypotheses

Volumn 67, Issue 5, 2006, Pages 1252-

  Wu, Xiong Zhi ^a  

^a TIANJIN MEDICAL UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

VASCULOTROPIN;

ANGIOGENESIS; BONE MARROW CELL; CARCINOGENESIS; CLEAR CELL CARCINOMA; GENE EXPRESSION; GENE MUTATION; HEMANGIOBLASTOMA; HEMATOPOIETIC STEM CELL; HUMAN; HYPOXIA; KIDNEY CANCER; KIDNEY CARCINOMA; LETTER; PHEOCHROMOCYTOMA; PRIORITY JOURNAL; VON HIPPEL LINDAU DISEASE;

ANOXIA; BONE MARROW CELLS; ENERGY METABOLISM; HUMANS; MUTATION; NEOPLASMS; NEOVASCULARIZATION, PATHOLOGIC; VASCULAR ENDOTHELIAL GROWTH FACTOR A; VON HIPPEL-LINDAU DISEASE;

EID: 33748038051     PISSN: 03069877     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.mehy.2006.03.043     Document Type: Letter

References (5)

1. Sharifi N., and Farrar W.L. Perturbations in hypoxia detection: A shared link between hereditary and sporadic tumor formation?. Med Hypotheses 66 (2006) 732-735
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4. von Marschall Z., Cramer T., Hocker M., Finkenzeller G., Wiedenmann B., and Rosewicz S. Dual mechanism of vascular endothelial growth factor upregulation by hypoxia in human hepatocellular carcinoma. Gut 48 (2001) 87-96
5. Kaplan R.N., Riba R.D., Zacharoulis S., Bramley A.H., Vincent L., and Costa C. VEGFR1-positive haematopoietic bone marrow progenitors initiate the pre-metastatic niche. Nature 438 (2005) 820-827