Adult mice lacking the p53/p63 target gene Perp are not predisposed to spontaneous tumorigenesis but display features of ectodermal dysplasia syndromes [2]

Cell Death and Differentiation

Volumn 13, Issue 9, 2006, Pages 1614-1618

  Ihrie, R A ^a   Bronson, R T ^b   Attardi, L D ^a  

^a STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b TUFTS UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CELL PROTEIN; DNA; PROTEIN P53; PROTEIN P63; PROTEIN PERP; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; CANCER SURVIVAL; CANCER SUSCEPTIBILITY; CARCINOGENESIS; CELL ADHESION; CONTROLLED STUDY; DESMOSOME; DNA DAMAGE; ECTODERMAL DYSPLASIA; GENE MUTATION; GENE TARGETING; GENETIC ANALYSIS; GENETIC TRANSCRIPTION; GENOTYPE; HETEROZYGOSITY; HETEROZYGOSITY LOSS; LETTER; LIVER CELL CARCINOMA; LUNG ADENOCARCINOMA; MOUSE; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; SKIN APPENDAGE; SKIN MALFORMATION; SOUTHERN BLOTTING;

AGING; ANIMALS; DISEASE SUSCEPTIBILITY; ECTODERMAL DYSPLASIA; EPITHELIUM; HAIR; HETEROZYGOTE; MEMBRANE PROTEINS; MICE; MICE, KNOCKOUT; NEOPLASMS; PHOSPHOPROTEINS; SKIN; TRANS-ACTIVATORS; TUMOR SUPPRESSOR PROTEIN P53;

EID: 33747085289     PISSN: 13509047     EISSN: 14765403     Source Type: Journal    
DOI: 10.1038/sj.cdd.4401871     Document Type: Letter

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