Estimation of effect of lipoic acid in the lung homogenates in rats during lipopolisaccharide-induced oxidative stress;Ocena działania kwasu liponowego w płucach szczura w czasie stresu oksydacyjnego wywołanego lipopolisacharydem

Polski Merkuriusz Lekarski

Volumn 20, Issue 120, 2006, Pages 651-654

  Skibska, Beata ^a   Ciejka, Elzbieta ^a   Gora̧ca, Anna ^a  

^a MEDICAL UNIVERSITY OF LODZ   (Poland)

Author keywords

Lipoic acid; Lipopolysaccharide; Oxidative stress

Indexed keywords

HYDROGEN PEROXIDE; LIPID; LIPOPOLYSACCHARIDE; SODIUM CHLORIDE; THIOBARBITURIC ACID REACTIVE SUBSTANCE; THIOCTIC ACID; THIOL GROUP;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONCENTRATION (PARAMETERS); CONTROLLED STUDY; DRUG EFFECT; LIPID PEROXIDATION; LUNG HOMOGENATE; MALE; NONHUMAN; OXIDATIVE STRESS; RAT;

ANIMALS; LIPID PEROXIDATION; LIPOPOLYSACCHARIDES; LUNG; MALE; OXIDATIVE STRESS; RATS; RATS, WISTAR; THIOCTIC ACID;

EID: 33746168224     PISSN: 14269686     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (25)

1. Antczak A., Nowak D., Shariati B. i wsp.: Increased hydrogen peroxide and thiobarbituric acid-reactive products in expired breath condensate of asthmatic patients. Eur. Respir. J., 1997, 10, 1235-1241.
2. Anuradha B., Varalakshmi P.: Protective role of DL-α-lipoic acid against mercury-induced neural lipid peroxidation. Pharmacol. Res., 1999, 39, 67-81.
3. Ayala A., Chaudry I.H.: Immune dysfunction in murine polimicrobial sepsis: mediators, macrophages, lymphocytes and apoptosis. Shock, 1996, 6, suppl. 1, 27-38.
4. Blackwell T.S., Yull F.E., Chen C.L. i wsp.: Multiorganic nuclear factor kappa B activation in a transgenic mouse model of systemic inflammation. Am. J. Respir. Crit. Care. Med., 2000, 162, 1095-101.
5. Coleman M.D., Eason R.C., Bailey C.J.: The therapeutic use of lipoic acid in diabetes: a current perspective. Environ. Toxicol. Pharmacol., 2001, 10, 167-172.
6. Ellman G.L.: SH groups determination in biological fluids. Anal. Biochem., 1970, 46, 237.
7. Glauser M.P., Zanetti G., Baumgartner J.D. i wsp.: Septic shock: pathogenesis. Lancet, 1991, 338, 732-736.
8. Granot E., Kohen R.: Oxidative stress in childhood - in health and disease states. Clinical Nutrition, 2004, 23, 3-11.
9. Han D., Handelman G., Marcocci L. i wsp.: Lipoic acid increases de novo synthesis of cellular glutathione by improving cysteine utilization. Biofactors, 1997, 6, 321-338.
10. Ikegami K., Lalonde C., Young Y.K. i wsp.: Comparison of plasma reduced glutathione and oxidized glutathione with lung and liver tissue oxidant activity during acute inflammation. Shock, 1994, 1, 307-312.
11. Kasielski M., Nowak D.: Long-term administration of N-acetylcysteine decreases hydrogen peroxide exhalation in subjects with chronic obstructive pulmonary disease. Respir. Med., 2001, 95, 448-456.
12. Khanna S., Atalay M., Laaksonen D.E. i wsp.: Alpha-lipoic acid supplementation: tissue glutathione homeostasis at rest and after exercise. J. Appl. Physiol. 1999, 86, 1191-1196.
13. Laskin D.L., Pendino K.J.: Macrophages and inflammatory mediators in tissue injury. Annu. Rev. Pharmacol. Toxicol., 1995, 35, 655-677.
14. Lee I.M.: Antioxidant vitamins in the prevention of cancer. Proc. Assoc. Am. Physicians, 1999, 111, 10-15.
15. Lowry O.H., Rosebrough N.J., Farr A.L. i wsp.: Protein measurements with Polin phenol reagent. J. Biol. Chem., 1951, 193, 265-275.
16. Packer L., Witt E.H., Tritschler H.J.: Alpha-Lipoic acid as a biological antioxidant. Free. Radic. Biol. Med., 1995, 19, 227-228.
17. 2 production by macrophages and neurotrophils with homovanilic acid and horse-radish peroxidase. J. Immunoi. Meth., 1983, 63, 347-357.
18. Savitha S., Tamilselvan J., Anusuyadevi M. i wsp.: Oxidative stress on mitochondrial antioxidant defense system in the aging process: Role of DL-α-lipoic acid and L-carnitine. Clin. Chim. Acta, 2005, 355, 173-180.
19. Selvakumar E., Prahalathan Ch., Mythili Y. i wsp.: Beneficial effects of DL-L-lipoic acid on cyclophosphamide-induced oxidative stress in mitochondrial fractions of rat testis. Chem. Biol. Interact., 2005, 152, 59-66.
20. Tatarková Z., Kaplán P., Matejov̌icová M. i wsp.: Effect of ischemia and reperfusion on protein oxidation in isolated rabbit hearts. Physiol. Res., 2005, 54, 185-191.
21. Thijs L.G., Groeneveld A.B., Hack C.E.: Bacterial endotoxin: Significance and pathogenesis of septic shock. w: Pathology of septic shock. Eds.: Rietschel ET, Wagner H. Springer-Verlag, Berlin-Heidelberg-New York, 1996, 209-237.
22. Victor V.M., De la Fuente M: Several functions of immune cells in mice changed by oxidative stress caused by endotoxin. Physiol. Res., 2003, 52, 789-796.
23. Victor V.M., Rocha M., De la Fuente M: Immune cells: free radicals and antioxidants in sepsis. Int. Immunopharmacol., 2004, 4, 327-347.
24. Ward P.A., Till G.O., Hatherill J.R. i wsp.: Systemic complement activation, lung injury, and products of lipid peroxidation. J. Clin. Ivest., 1985, 76, 517-527.
25. Wilson M.E.: Effect of bacterial endotoxins on neutrophil function. Rev. Infect. Dis., 1985, 7, 404-418.