Innate immune tissue injury and murine HGA: tissue injury in the murine model of granulocytic anaplasmosis relates to host innate immune response and not pathogen load.

Annals of the New York Academy of Sciences

Volumn 1063, Issue , 2005, Pages 425-428

  Scorpio, Diana G ^a   Von Loewenich, Friederike D ^a   Bogdan, Christian ^a   Dumler, J Stephen ^a  

^a JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

INDUCIBLE NITRIC OXIDE SYNTHASE; NOS2 PROTEIN, MOUSE; TUMOR NECROSIS FACTOR;

ANAPLASMA PHAGOCYTOPHILUM; ANAPLASMOSIS; ANIMAL; ARTICLE; C57BL MOUSE; DISEASE MODEL; GENETICS; HUMAN; IMMUNOLOGY; INNATE IMMUNITY; METABOLISM; MICROBIOLOGY; MOUSE; MOUSE MUTANT; PATHOLOGY;

ANAPLASMA PHAGOCYTOPHILUM; ANAPLASMOSIS; ANIMALS; DISEASE MODELS, ANIMAL; HUMANS; IMMUNITY, NATURAL; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MICE, SCID; NITRIC OXIDE SYNTHASE TYPE II; TUMOR NECROSIS FACTORS;

EID: 33745727538     PISSN: 00778923     EISSN: None     Source Type: Journal    
DOI: 10.1196/annals.1355.077     Document Type: Article

References (0)