Human polyomavirus JC and BK persistent infection

Advances in Experimental Medicine and Biology

Volumn 577, Issue , 2006, Pages 102-116

  Doerries, Kristina ^a  

^a UNIVERSITY OF WÜRZBURG   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

VIRUS DNA;

BK VIRUS; BLOOD; GENETICS; HUMAN; IMMUNOLOGY; JC VIRUS; METABOLISM; PATHOGENICITY; PATHOPHYSIOLOGY; REVIEW; TISSUE DISTRIBUTION; VIROLOGY; VIRUS INFECTION;

BK VIRUS; DNA, VIRAL; HUMANS; JC VIRUS; POLYOMAVIRUS INFECTIONS; TISSUE DISTRIBUTION;

EID: 33745599157     PISSN: 00652598     EISSN: None     Source Type: Book Series    
DOI: 10.1007/0-387-32957-9_8     Document Type: Review

References (13)

1. Hirsch HH, Steiger J. Polyomavirus BK. Important review on BKV virology and pathogenesis. Lancet Infect Dis 2003; 3:611-623.
2. Walker DL, Padgett BL. The classical summary on PML. Progressive multifocal leukoencephalopathy. New York: Plenum Press, 1983; 18:161-193.
3. Doerries K, Sbiera S, Drews K et al. A study on JCV affected peripheral blood cell subopopulations emphasizing granulocytes as major affected cell type. Association of human polyomavirus JC with peripheral blood of immunoimpaired and healthy individuals. J Neurovirol 2003; 9(Suppl 1):81-87.
4. Arthur RR, Shah KV. Occurrence and significance of papovaviruses BK and JC in the urine. A summary of early studies of JCV and BKV viruria that remains a valuable source for the understanding of persistent huPyV infections. Prog Med Virol 1989; 36:42-61.
5. Doerries K. Latent and persistent human polyomavirus infection. Extensive review on persistent polyomavirus infection. In: Khalili K, Stoner GL, eds. Human Polyomaviruses. Molecular and clinical perspectives. New York: Wiley-Liss Inc. 2001:197-237.
6. Koralnik IJ, Boden D, Mai VX et al. JC virus DNA load in patients with and without progressive multifocal leukoencephalopathy. Important paper on the presence of JCV in different target compartments. Neurology 1999a; 52(2):253-260.
7. Du Pasquier RA, Corey S, Margolin DH et al. Productive infection of cerebellar granule cell neurons by JC virus in an HIV+ individual. The first report on the involvement of neurons in JCV infection. Neurology 2003; 61:775-782.
8. von Einsiedel RW, Samorei IW, Pawlita M et al. New JC virus infection patterns by in situ polymerase chain reaction in brains of acquired immunodeficiency syndrome patients with progressive multifocal leukoencephalopathy. Characterization of affected cell types and topography of PML lesions by modern molecular methods. J Neurovirol 2004; 10:1-11.
9. Doerries K. Human polyomaviruses in target cells of hematopoietic origin and mechanisms of activation in the course of infection. The review concentrates on interaction of huPyV with cells of hematopoietic origin. Current topics in Virology 1999b; 1:84-93.
10. Jensen PN, Major EO. Viral variant nucleotide sequences help expose leukocytic positioning in the JC virus pathway to the CNS. Concentrates on the possible role of JCV variants for viral dissemination. J Leukoc Biol 1999; 65:428-438.
11. Koralnik IJ, Schmitz JE, Lifton MA et al. Detection of JC virus DNA in peripheral blood cell subpopulations of HIV-1-infected individuals. Characterization of PBC subpopulations involved in JCV infection. J Neurovirol 1999b; 5:430-435.
12. Wei G, Liu CK, Atwood WJ. JC virus binds to primary human glial cells, tonsillar stromal cells, and B-lymphocytes, but not to T lymphocytes. Addresses the role of transcription factors and cell receptors for JCV infection. J Neurovirology 2000; 6:127-136.
13. Monaco MC, Sabath BF, Durham LC et al. JC virus multiplication in human hematopoietic progenitor cells requires the NF-1 class D transcription factor. Dependence of JCV replication on transcription factor family NF expression. J Virol 2001; 75:9687-9695.