The role of extracellular matrix remodeling in carcinogenesis and metastasis

Medical Hypotheses

Volumn 67, Issue 2, 2006, Pages 425-

  Wu, Xiong Zhi ^a  

^a TIANJIN MEDICAL UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

CELL ADHESION MOLECULE; FIBRONECTIN; VASCULOTROPIN RECEPTOR 1; VERY LATE ACTIVATION ANTIGEN 4;

CARCINOGENESIS; CELL FUSION; CELL PROLIFERATION; EXTRACELLULAR MATRIX; HEMATOPOIETIC STEM CELL; HYPOTHESIS; LETTER; METASTASIS; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN EXPRESSION;

CELL TRANSFORMATION, NEOPLASTIC; EXTRACELLULAR MATRIX; HUMANS; NEOPLASM METASTASIS;

EID: 33744918048     PISSN: 03069877     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.mehy.2006.03.020     Document Type: Letter

References (3)

1. Parris G.E. The cell clone ecology hypothesis and the cell fusion model of cancer progression and metastasis (II): three pathways for spontaneous cell-cell fusion and escape from the intercellular matrix. Med Hypotheses 67 (2006) 172-176 10.1016/j.mehy. 2006.01.018
2. Wu X.Z., Chen D., and Xie G.R. Extracellular matrix remodeling in hepatocellular carcinoma: effects of soil on seed?. Med Hypotheses 66 (2006) 1115-1120
3. Kaplan R.N., Riba R.D., Zacharoulis S., et al. VEGFR1-positive haematopoietic bone marrow progenitors initiate the pre-metastatic niche. Nature 438 (2005) 820-827