-
1
-
-
0034727833
-
Genomic aberrations and survival in chronic lymphocytic leukemia
-
Dohner H., Stilgenbauer S., Benner A., Leupolt E., Krober A., Bullinger L., Dohner K., Bentz M., and Lichter P. Genomic aberrations and survival in chronic lymphocytic leukemia. N Engl J Med 343 (2000) 1910-1916
-
(2000)
N Engl J Med
, vol.343
, pp. 1910-1916
-
-
Dohner, H.1
Stilgenbauer, S.2
Benner, A.3
Leupolt, E.4
Krober, A.5
Bullinger, L.6
Dohner, K.7
Bentz, M.8
Lichter, P.9
-
2
-
-
0037541558
-
Chromosome anomalies detected by interphase fluorescence in situ hybridization: correlation with significant biological features of B-cell chronic lymphocytic leukaemia
-
Dewald G.W., Brockman S.R., Paternoster S.F., Bone N.D., O'Fallon J.R., Allmer C., James C.D., Jelinek D.F., Tschumper R.C., Hanson C.A., Pruthi R.K., Witzig T.E., Call T.G., and Kay N.E. Chromosome anomalies detected by interphase fluorescence in situ hybridization: correlation with significant biological features of B-cell chronic lymphocytic leukaemia. Br J Haematol 121 (2003) 287-295
-
(2003)
Br J Haematol
, vol.121
, pp. 287-295
-
-
Dewald, G.W.1
Brockman, S.R.2
Paternoster, S.F.3
Bone, N.D.4
O'Fallon, J.R.5
Allmer, C.6
James, C.D.7
Jelinek, D.F.8
Tschumper, R.C.9
Hanson, C.A.10
Pruthi, R.K.11
Witzig, T.E.12
Call, T.G.13
Kay, N.E.14
-
3
-
-
0025319412
-
Chromosome aberrations in B-cell chronic lymphocytic leukemia. Pathogenetic and clinical implications
-
Juliusson G., and Gahrton G. Chromosome aberrations in B-cell chronic lymphocytic leukemia. Pathogenetic and clinical implications. Cancer Genet Cytogenet 45 (1990) 143-160
-
(1990)
Cancer Genet Cytogenet
, vol.45
, pp. 143-160
-
-
Juliusson, G.1
Gahrton, G.2
-
4
-
-
0025112785
-
Prognostic subgroups in B-cell chronic lymphocytic leukemia defined by specific chromosomal abnormalities
-
Juliusson G., Oscier D.G., Fitchett M., Ross F.M., Stockdill G., Mackie M.J., Parker A.C., Castoldi G.L., Guneo A., Knuutila S., et al. Prognostic subgroups in B-cell chronic lymphocytic leukemia defined by specific chromosomal abnormalities. N Engl J Med 323 (1990) 720-724
-
(1990)
N Engl J Med
, vol.323
, pp. 720-724
-
-
Juliusson, G.1
Oscier, D.G.2
Fitchett, M.3
Ross, F.M.4
Stockdill, G.5
Mackie, M.J.6
Parker, A.C.7
Castoldi, G.L.8
Guneo, A.9
Knuutila, S.10
-
5
-
-
0033034241
-
Mapping of the breakpoints on the short arm of chromosome 17 in neoplasms with an i(17q)
-
Scheurlen W.G., Schwabe G.C., Seranski P., Joos S., Harbott J., Metzke S., Dohner H., Poustka A., Wilgenbus K., and Haas O.A. Mapping of the breakpoints on the short arm of chromosome 17 in neoplasms with an i(17q). Genes Chromosomes Cancer 25 (1999) 230-240
-
(1999)
Genes Chromosomes Cancer
, vol.25
, pp. 230-240
-
-
Scheurlen, W.G.1
Schwabe, G.C.2
Seranski, P.3
Joos, S.4
Harbott, J.5
Metzke, S.6
Dohner, H.7
Poustka, A.8
Wilgenbus, K.9
Haas, O.A.10
-
6
-
-
9144264835
-
The breakpoint region of the most common isochromosome, i(17q), in human neoplasia is characterized by a complex genomic architecture with large, palindromic, low-copy repeats
-
Barbouti A., Stankiewicz P., Nusbaum C., Cuomo C., Cook A., Hoglund M., Johansson B., Hagemeijer A., Park S.S., Mitelman F., Lupski J.R., and Fioretos T. The breakpoint region of the most common isochromosome, i(17q), in human neoplasia is characterized by a complex genomic architecture with large, palindromic, low-copy repeats. Am J Hum Genet 74 (2004) 1-10
-
(2004)
Am J Hum Genet
, vol.74
, pp. 1-10
-
-
Barbouti, A.1
Stankiewicz, P.2
Nusbaum, C.3
Cuomo, C.4
Cook, A.5
Hoglund, M.6
Johansson, B.7
Hagemeijer, A.8
Park, S.S.9
Mitelman, F.10
Lupski, J.R.11
Fioretos, T.12
-
7
-
-
0345299213
-
Isochromosome 17q in blast crisis of chronic myeloid leukemia and in other hematologic malignancies is the result of clustered breakpoints in 17p11 and is not associated with coding TP53 mutations
-
Fioretos T., Strombeck B., Sandberg T., Johansson B., Billstrom R., Borg A., Nilsson P.G., Van Den Berghe H., Hagemeijer A., Mitelman F., and Hoglund M. Isochromosome 17q in blast crisis of chronic myeloid leukemia and in other hematologic malignancies is the result of clustered breakpoints in 17p11 and is not associated with coding TP53 mutations. Blood 94 (1999) 225-232
-
(1999)
Blood
, vol.94
, pp. 225-232
-
-
Fioretos, T.1
Strombeck, B.2
Sandberg, T.3
Johansson, B.4
Billstrom, R.5
Borg, A.6
Nilsson, P.G.7
Van Den Berghe, H.8
Hagemeijer, A.9
Mitelman, F.10
Hoglund, M.11
-
8
-
-
0029079152
-
Comparative genomic hybridization in chronic B-cell leukemias shows a high incidence of chromosomal gains and losses
-
Bentz M., Huck K., du Manoir S., Joos S., Werner C.A., Fischer K., Dohner H., and Lichter P. Comparative genomic hybridization in chronic B-cell leukemias shows a high incidence of chromosomal gains and losses. Blood 85 (1995) 3610-3618
-
(1995)
Blood
, vol.85
, pp. 3610-3618
-
-
Bentz, M.1
Huck, K.2
du Manoir, S.3
Joos, S.4
Werner, C.A.5
Fischer, K.6
Dohner, H.7
Lichter, P.8
-
9
-
-
0036468807
-
Genome architecture, rearrangements and genomic disorders
-
Stankiewicz P., and Lupski J.R. Genome architecture, rearrangements and genomic disorders. Trends Genet 18 (2002) 74-82
-
(2002)
Trends Genet
, vol.18
, pp. 74-82
-
-
Stankiewicz, P.1
Lupski, J.R.2
-
10
-
-
0031731487
-
Genomic disorders: structural features of the genome can lead to DNA rearrangements and human disease traits
-
Lupski J.R. Genomic disorders: structural features of the genome can lead to DNA rearrangements and human disease traits. Trends Genet 14 (1998) 417-422
-
(1998)
Trends Genet
, vol.14
, pp. 417-422
-
-
Lupski, J.R.1
-
11
-
-
0038067849
-
Genome architecture catalyzes nonrecurrent chromosomal rearrangements
-
Stankiewicz P., Shaw C.J., Dapper J.D., Wakui K., Shaffer L.G., Withers M., Elizondo L., Park S.S., and Lupski J.R. Genome architecture catalyzes nonrecurrent chromosomal rearrangements. Am J Hum Genet 72 (2003) 1101-1116
-
(2003)
Am J Hum Genet
, vol.72
, pp. 1101-1116
-
-
Stankiewicz, P.1
Shaw, C.J.2
Dapper, J.D.3
Wakui, K.4
Shaffer, L.G.5
Withers, M.6
Elizondo, L.7
Park, S.S.8
Lupski, J.R.9
-
12
-
-
3242741800
-
Genomic disorders-genome architecture results in suspectibility to DNA rearrangements causing common human traits
-
Stankiewicz P., Inoue K., Bi W., Walz W., Park S.S., Kurotaki N., Shaw C.J., Fonseca P., Yan J., Lee J.A., Khajav M., and Lupski J.R. Genomic disorders-genome architecture results in suspectibility to DNA rearrangements causing common human traits. Cold Spring Harbor Symp Quant Biol 68 (2003) 445-454
-
(2003)
Cold Spring Harbor Symp Quant Biol
, vol.68
, pp. 445-454
-
-
Stankiewicz, P.1
Inoue, K.2
Bi, W.3
Walz, W.4
Park, S.S.5
Kurotaki, N.6
Shaw, C.J.7
Fonseca, P.8
Yan, J.9
Lee, J.A.10
Khajav, M.11
Lupski, J.R.12
-
13
-
-
0037162516
-
A 76-kb duplicon maps close to the BCR gene on chromosome 22 and the ABL gene on chromosome 9: possible involvement in the genesis of the Philadelphia chromosome translocation
-
Saglio G., Storlazzi C.T., Giugliano E., Surace C., Anelli L., Rege-Cambrin G., Zagaria A., Jimenez Velasco A., Heiniger A., Scaravaglio P., Torres Gomez A., Roman Gomez J., Archidiacono N., Banfi S., and Rocchi M. A 76-kb duplicon maps close to the BCR gene on chromosome 22 and the ABL gene on chromosome 9: possible involvement in the genesis of the Philadelphia chromosome translocation. Proc Natl Acad Sci USA 99 (2002) 9882-9887
-
(2002)
Proc Natl Acad Sci USA
, vol.99
, pp. 9882-9887
-
-
Saglio, G.1
Storlazzi, C.T.2
Giugliano, E.3
Surace, C.4
Anelli, L.5
Rege-Cambrin, G.6
Zagaria, A.7
Jimenez Velasco, A.8
Heiniger, A.9
Scaravaglio, P.10
Torres Gomez, A.11
Roman Gomez, J.12
Archidiacono, N.13
Banfi, S.14
Rocchi, M.15
-
15
-
-
0034141541
-
Immunostimulatory CpG-oligonucleotides cause proliferation, cytokine production, and an immunogenic phenotype in chronic lymphocytic leukemia B cells
-
Decker T., Schneller F., Sparwasser T., Tretter T., Lipford G.B., Wagner H., and Peschel C. Immunostimulatory CpG-oligonucleotides cause proliferation, cytokine production, and an immunogenic phenotype in chronic lymphocytic leukemia B cells. Blood 95 (2000) 999-1006
-
(2000)
Blood
, vol.95
, pp. 999-1006
-
-
Decker, T.1
Schneller, F.2
Sparwasser, T.3
Tretter, T.4
Lipford, G.B.5
Wagner, H.6
Peschel, C.7
-
16
-
-
0036047670
-
CD40L stimulation enhances the ability of conventional metaphase cytogenetics to detect chromosome aberrations in B-cell chronic lymphocytic leukaemia cells
-
Buhmann R., Kurzeder C., Rehklau J., Westhaus D., Bursch S., Hiddemann W., Haferlach T., Hallek M., and Schoch C. CD40L stimulation enhances the ability of conventional metaphase cytogenetics to detect chromosome aberrations in B-cell chronic lymphocytic leukaemia cells. Br J Haematol 118 (2002) 968-975
-
(2002)
Br J Haematol
, vol.118
, pp. 968-975
-
-
Buhmann, R.1
Kurzeder, C.2
Rehklau, J.3
Westhaus, D.4
Bursch, S.5
Hiddemann, W.6
Haferlach, T.7
Hallek, M.8
Schoch, C.9
-
17
-
-
30444451801
-
Chromosomal translocations are associated with poor prognosis in chronic lymphocytic leukemia
-
Mayr C., Speicher M.R., Kofler D.M., Buhmann R., Strehl J., Busch R., Hallek M., and Wendtner C.M. Chromosomal translocations are associated with poor prognosis in chronic lymphocytic leukemia. Blood 107 (2006) 742-751
-
(2006)
Blood
, vol.107
, pp. 742-751
-
-
Mayr, C.1
Speicher, M.R.2
Kofler, D.M.3
Buhmann, R.4
Strehl, J.5
Busch, R.6
Hallek, M.7
Wendtner, C.M.8
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