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Volumn 100, Issue 5, 2006, Pages 1437-1438
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Possible new mechanism underlying hypertonic saline therapy for cerebral edema
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Author keywords
[No Author keywords available]
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Indexed keywords
ADENOSINE TRIPHOSPHATE;
ARGIPRESSIN;
ARGIPRESSIN RECEPTOR;
CHLORIDE ION;
COTRANSPORTER;
MANNITOL;
NEUROPEPTIDE;
OSMOTIC AGENT;
POTASSIUM CHANNEL;
POTASSIUM ION;
SODIUM CHLORIDE;
ABSENCE OF SIDE EFFECTS;
BLOOD BRAIN BARRIER;
BLOOD VISCOSITY;
BRAIN EDEMA;
BRAIN ISCHEMIA;
CLINICAL PRACTICE;
DISEASE CONTROL;
DRUG EFFICACY;
DRUG MECHANISM;
DRUG SAFETY;
EDITORIAL;
HEMATOCRIT;
HUMAN;
HYPEROSMOLARITY;
INTRACELLULAR FLUID;
MIDDLE CEREBRAL ARTERY;
NONHUMAN;
OSMOSIS;
PLASMA OSMOLARITY;
PRIORITY JOURNAL;
REBOUND;
REPERFUSION;
ANIMALS;
ARGININE VASOPRESSIN;
BRAIN EDEMA;
DISEASE MODELS, ANIMAL;
HUMANS;
ISCHEMIC ATTACK, TRANSIENT;
MALE;
MANNITOL;
RATS;
SALINE SOLUTION, HYPERTONIC;
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EID: 33646420058
PISSN: 87507587
EISSN: 15221601
Source Type: Journal
DOI: 10.1152/japplphysiol.01560.2005 Document Type: Editorial |
Times cited : (3)
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References (10)
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