Corticosterone regulates expression of CCL2 in the intact and chemically injured hippocampus

Neuroscience Letters

Volumn 399, Issue 1-2, 2006, Pages 162-166

  Little, Alvin R ^a   Sriram, Krishnan ^a   O'Callaghan, James P ^a  

^a NATIONAL INSTITUTE FOR OCCUPATIONAL SAFETY AND HEALTH   (United States)

Author keywords

Brain; CCL2; Corticosterone; Glucocorticoids; MCP 1; Microglia; Neurodegeneration; Neuroinflammation; Trimethyltin

Indexed keywords

ALPHA INTEGRIN; CHEMOKINE RECEPTOR; CHEMOKINE RECEPTOR CCR2; CHEMOKINE RECEPTOR CCR5; CORTICOSTERONE; GLUCOCORTICOID; INTERLEUKIN 1ALPHA; MONOCYTE CHEMOTACTIC PROTEIN 1; TRIMETHYLTIN;

ADRENALECTOMY; ANALYTIC METHOD; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL ACTIVATION; CHEMICAL INJURY; CONTROLLED STUDY; CYTOKINE PRODUCTION; GENE EXPRESSION REGULATION; HIPPOCAMPUS; MALE; MICROGLIA; NERVE DEGENERATION; NONHUMAN; PRIORITY JOURNAL; RAT;

ADRENALECTOMY; ANIMALS; ANTIGENS, CD11B; ASTROCYTES; CHEMOKINE CCL2; CORTICOSTERONE; GLIOSIS; GLUCOCORTICOIDS; HIPPOCAMPUS; IMMUNOSUPPRESSIVE AGENTS; MALE; NEURONS; RATS; RATS, LONG-EVANS; RECEPTORS, CCR5; RECEPTORS, CHEMOKINE; RNA, MESSENGER; TRIMETHYLTIN COMPOUNDS;

EID: 33646086971     PISSN: 03043940     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neulet.2006.01.050     Document Type: Article

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