Adventitious Mutations in Clinical Grade Vectors: An Issue to Consider?

Molecular Therapy

Volumn 13, Issue 5, 2006, Pages 831-832

  Williams, David A ^a   Cripe, Timothy P ^a  

^a UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ONCOLYTIC VIRUS; VIRUS VECTOR;

BACTERIAL ARTIFICIAL CHROMOSOME; CANCER GROWTH; EDITORIAL; GENE SEQUENCE; HERPES SIMPLEX VIRUS 1; HUMAN; NONSENSE MUTATION; VIRAL GENE DELIVERY SYSTEM; VIRAL GENE THERAPY; VIRUS GENOME; VIRUS MUTATION;

BACTERIA (MICROORGANISMS); HUMAN HERPESVIRUS 1;

EID: 33645853165     PISSN: 15250016     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.ymthe.2005.03.040     Document Type: Editorial

References (3)

1. M.J. Dambach Oncolytic viruses derived from the g34.5-deleted Herpes simplex virus R3616 encode a truncated UL3 protein Mol. Ther. 13 2006 this issue.
2. K. Terada Development of a rapid method to generate multiple oncolytic HSV vectors and their in vivo evaluation using syngeneic mouse tumor models Gene Ther. 2006 (in the press).
3. D.A. Williams An Emerging Consensus on Recommendations to Facilitate Clinical Gene Transfer Mol. Ther. 13 2006 637 638